 Synonyms  Secretory otitis media  Non- purulent otitis media  Serous otitis media  Glue ear  Definition: presence of non-purulent fluid within.

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Presentation transcript:

 Synonyms  Secretory otitis media  Non- purulent otitis media  Serous otitis media  Glue ear  Definition: presence of non-purulent fluid within middle ear cleft. Acute or chronic(more than 12 weeks).

Otitis Media  It is the most common disease of childhood, next to viral URTI.  It is acute bacterial infection in 80% (1-6 years)  The most frequent disease treated with antibiotics.

Incidence  Children more than adults, peak age:5-6 years  Male more than female  Winter months

Aetiology  1- Eustachian tube dysfunction ET has 2 main functions: 1- Regulate ME pressure at atmospheric level. 2- Drainage of ME fluid into nasopharynx. ET dysfunction may be due to: A- Up. respiratory tract infection B- Allergy C- Cilial abnormality as Kartageners syndrome. Children have poorer ET function than adults.

 2- Cleft palate : muscles which open the ET attached to the soft palate (tensor and levator palate muscles).  3- Adenoid hypertrophy: causes obstruction of ET orifice in the nasopharynx which leads to negative ME pressure and hence ME effusion.  4- Nasopharyngeal tumours: similar mechanism to adenoid hypertrophy.  5- Radiotherapy to neck  6- Idiopathic  7- Barotrauma :as during airplane descent  8- AIDS

Clinical features  1- Deafness : the chief complaint  2- Impaired speech and language development ( in children)  3- Behavioral deterioration and scholastic difficulties (in children)  4- Pain is rare unless there is superadded infection  5- Tinnitus and vertigo

Normal Ear Drum

Serous Otitis Media

On examination of the tympanic membrane by otoscopy:  Retraction  Loss of translucency  Color changes: from pale grey to bluish black  Fluid level or air bubbles  Splitting or absent light reflex  Displacement of malleus handle and it become more prominent. Tuning fork tests:  Rinne test :negative(bone conduction better than air)  Weber test: lateralized to diseased side Audiometry:  Pure tone audiometry: air-bone gap  Tympanometry: flat type B curve.

OTITIS MEDIA WITH EFFUSION Diagnosis PLAY AUDIOMETRY AUDIOMETRY TYMPANOMETRY

Treatment High rate of spontaneous resolution. Only or 5% persist more than 1 year. Medical treatment  A- antibiotics: days course  B- antihistamines  C- nasal decongestant  D- steroids: local in the nose or oral short course.  E- autoinflation of the ear by Valsalva maneuver. Surgical treatment:  If persist more than 12 weeks with no benefit to medical treatment:  A- myringotomy with or without tympanostomy tube insertion(grommet)  B- adenoidectomy.  C- myringotomy with adenoidectomy  D- cortical mastoidectomy

OTITIS MEDIA WITH EFFUSION Treatment Adeno – Tonsellectomy&Myringotomy tube insertion (T&A &TUBES)

Otosclerosis Definition : Hereditary ear disease in which the mature bone is replaced by a woven bone of more cellularity. Site : Oval window region is the commonest site of involvement thus leading to fixation of stapes and conductive hearing impairment.

 Incidence : Age :peak years Gender : female more than male Increased in severity during pregnancy and menopause.  Clinical features: 1- deafness : the main symptom, gradual, conductive in early stages, then mixed conductive and sensorineural. Bilateral 2- tinnitus 3- vertigo On examination: normal tympanic membrane color and mobility, but there may be red flush on the tympanic membrane called flamingo flush or Schwartz sign.  Tuning fork test : Rinne negative and Weber lateralized to affected side.  Audiometry: pure tone audiometry shows air-bone gap and Tympanometry shows normal or shallow curve type As.

Differential diagnosis : other causes of conductive deafness:  1- otitis media with effusion  2- ossicular disconnection  3- ME fibrosis  4- congenital cholesteatoma  5- osteogenesis imperfecta Treatment :  1- medical  A- hearing aid  B- sodium fluoride  2- surgical : stapedectomy in which the stapes removed and replaced by a synthetic prosthesis under local or general anesthesia.

Menieres disease  Disease of inner ear in which there is hydropic distension of the endolymphatic system, characterized by episodic vertigo, deafness and tinnitus.  Endolymphatic distension(hydrops) could be idiopathic called menieres disease or secondary to chronic otitis media, syphilis, trauma, leukemia, metabolic disease, viral infection, autoimmunity or Otosclerosis.  Clinical features :  1- episodic vertigo associated with vegetative symptoms(nausea and vomiting),last for 2-3 hours, less than 24 hours (24min-24hr).  2- deafness: sensorineural, fluctuate and progressive.  3- tinnitus  4- aural fullness.

Treatment :  1- medical : A- reassurance B- labyrinthine sedative drugs(vestibular suppressants) :1- phenothiazines : as prochloperazine (stemetel) 2- antihistamines: as cinnarizine (stugeron) 3- benzodiazepines: diazepam and lorazepam C- prophylaxis: between the attacks: diuretics, salt restriction, vasodilators as betahistine (betaserc) and nitroglycerine D- steroids and cytotoxic drugs might be helpful E- hearing aids  2- surgical Only 10-20% need surgery A- endolymphatic sac decompression B- surgical or chemical ablation of vestibular nerve and vestibular end organs C- labyrinthectomy: total destruction of sensory cells of inner ear.