TESTIMONY TESTIMONY HEMATOMA INTRAPARENCHYMAL TEMPORO- OCCIPITAL DRA. ITHAMAR RODRÍGUEZ VENEZUELA DRA. ITHAMAR RODRÍGUEZ VENEZUELA And he said to me: My grace is sufficient for thee: for my strength is made perfect in weakness. Therefore most gladly I will rather boast in my infirmities, that rest upon me the power of Christ. " Why which, for Christ's sake I delight in weaknesses, because when I am weak then I am strong. " 2 Corinthians

CASE REPORT PATIENT: FEMALE / 53 YEARS DIAGNOSIS: BLEEDING INTRAPARENCHYMAL RIGHT-OCCIPITAL TEMPORO PATIENT IS PRESENT HEADACHE OF 3 DAYS OF EVOLUTION. PHENOMENON DEJAVU PROYECTILE VOMITING VISION LOSS ON 3 SEPTEMBER 2013 THE FEMALE PATIENT 53 YEARS COMES TO EMERGENCY WITH HEADACHE WITH 3 DAY OF EVOLUTION, VOMITING AND LOSS OF VISION.

CASE REPORT PATIENT: FEMALE / 53 YEARS BLOOD PRESSURE 120/80 mmHg PULSE: 90 EKG: AWARE HYDRATION POOR WITH VISION LOSS ID: 1. HEMORRHAGIC ACV. 2.HEMATOMA INTRAPARENCHYMAL TEMPORO-OCCIPITAL 3.LOE CEREBRAL s BACKGROUND: - NEGATIVE DIABETE E HYPERTENSION. CT of intracerebral hematoma reports 09/03/2013 temporooccipital law measures 30x57x25 mm with perilesional edema associated hyperdense + elongated transverse sinus thrombosis.

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ABOUT THE CAUSES: This evidence was rejected by neurosurgery department because it is extremely rare cases (0.5% Cerebrovascular enf.) TCF not conduct antithrombotic therapy indicated for being a bleeding intraparequimatosa 5.7 ​​ cm and volume expectantly to the resonance on December 5th and discussed by the specialties the December the 20th, 2013 Since I started treatment with clopidogrel and Asa and I / C with ongoing Hematology “SIGN OF THE CORD” "NO SIGNAL WITHIN RIGHT TRANSVERSE AND SUPERIOR LONGITUDINAL SINUS"

Testimony Dr. Ithamar Rodriguez. In meeting with the fasting group AMEC Zulia on December the 21st, 2013, I showed them the need for specific prayer recanalization of these still clogged cerebral veins, and the danger of increased intracerebral pressure with subsequent potential risks. Teaching preserve health achieved in Christ, for the presence of the Lord. "See, you are well, sin no more, do not come to something worse. " John: 5:14

CASE REPORT About causes: December 20th, 2013 is determined in discussion with neurosurgery and neuroradiology that the cause of this hematoma was due to cerebral venous thrombosis, which began in the superior sagittal sinus and extended into the transverse sinus and internal jugular right, causing intraparenchymal hemorrhage spontaneous.With the cerebral resonance of December the 5 th 2013, for obstruction of these sinuses and according Doppler Carotid, recanalization of 30% of the internal jugular, so new rear controls are required and expected higher recanalization. The cerebral venous thrombosis: TVC ( clots in cerebral veins and sinuses ) Described in the early twentieth century. Less than 0.5 % of strokes associated with prothrombotic states, with an annual incidence of 3-4 cases per 1,000,000 inhabitants. Rare condition can occur at any age, more common in women between 40s. -and 50s. decade of life in the absence of vascular risk factors. Pregnancy, puerperium, use of ACO. Or TRH., Dehydration, trauma, systemic diseases, neoplasms, collagen, blood dyscrasias, are among the causes.

CASE REPORT Clinical and radiological manifestations are diverse and difficult recognition so the diagnosis often goes unnoticed. Their treatment differs from other STROKE. The diagnosis is based on an index of suspicion and confirmation with neuroimaging studies. The magnetic resonance venography or venous Angiotac are the main diagnostic tests. Diagnosis should be made as soon as possible, as early anticoagulant therapy in over 80% present with good prognosis. You can have less acute course of three days, 30-day subacute evolution or chronic, more than 30 days. With a lower rate of mortality of 10% in patients treated promptly and more than 30% in untreated with a recurrence rate of 2 to 5% in the first year of the event. Factors of poor prognosis in early stage hemorrhagic infarction, male and older than 37 years are described.

CASE REPORT The clinic is due to intracranial hypertension, local edema with eventual secondary venous infarction and cerebral hemorrhage cerebral venous thrombosis. The most frequent symptoms are headache in 90%, seizures, papilledema and focal neurological defect depending on the affected vessels, disorders of consciousness. It must be timely, if prevented seizures and intracranial hypertension puts life at risk decompressive craniotomy practice. Tatar 1) the underlying causes that are detected as infections, autoimmune, or jugular catheterization, 2) antithrombotic treatment according to a recent metaanálisi has proven useful in more than 80% of 624 patients treated in the study of international TVC received anticoagulants 79% of favorable events despite hemorrágicos.Se used low molecular weight heparin and then vitamin K inhibitors for 6 months to a year according to risk of recurrence. Bibliography. Dr.Jesús Perez. Stand Ehlissham J. Stem.Mousser. Callejas. Medline.

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