Obesity resistance and increased hepatic expression of catabolism ‐ related mRNAs in Cnot3 +/− mice by Masahiro Morita, Yuichi Oike, Takeshi Nagashima, Tsuyoshi Kadomatsu, Mitsuhisa Tabata, Toru Suzuki, Takahisa Nakamura, Nobuaki Yoshida, Mariko Okada, and Tadashi Yamamoto EMBO J. Volume 30(22): November 16, 2011 ©2011 by European Molecular Biology Organization
Leanness and reduced lipid content in Cnot3+/− mice. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization
Increased glucose homeostasis, insulin sensitivity, and metabolic rates in Cnot3+/− mice. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization
Resistance to diet ‐ induced obesity and related metabolic disorder in Cnot3+/− mice. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization
Improvement of obesity and insulin resistance in ob/ob;Cnot3+/− mice. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization
Deadenylase regulates genes involved in metabolism in the liver. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization
CNOT3 reduction affects the poly(A) tail length of mRNAs involved in lipid metabolism and growth. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization
Alternation of CNOT3 expression level by responding to nutrient status and identification of CNOT3 ‐ regulated mRNAs. Masahiro Morita et al. EMBO J. 2011;30: ©2011 by European Molecular Biology Organization