Dr Samira Arami General Cardiologist
Conductive system
LEAD TYPEPOSITIVE INPUTNEGATIVE INPUT Standard Limb Leads Lead ILeft armRight arm Lead IILeft legRight arm Lead IIILeft legLeft arm Augmented Limb Leads aVRRight armLeft arm plus left leg aVLLeft armRight arm plus left leg aVFLeft legLeft arm plus right arm
Precordial Leads* V1V1 Right sternal margin, fourth intercostal space Wilson central terminal V2V2 Left sternal margin, fourth intercostal space Wilson central terminal V3V3 Midway between V 2 and V 4 Wilson central terminal V4V4 Left midclavicular line, 5th intercostal space Wilson central terminal V5V5 Left anterior axillary line [†] Wilson central terminal V6V6 Left midaxillary line [†] Wilson central terminal V7V7 Posterior axillary line [†] Wilson central terminal V8V8 Posterior scapular line [†] Wilson central terminal V9V9 Left border of spine [†] Wilson central terminal
ECG Leads Limb leads: I, II, III, aVR, aVL, aVF, Chest leads: V1-V6 Anterior surface: V1-4. Inferior surface: II, III and aVF. Lateral surface: I, aVL and V5-6.
ECG Paper
ECG Complex
WAVE OR INTERVAL DURATION (msec) P wave duration<120 PR interval (3-5mm) QRS duration< (2.5-3mm) QT interval (corrected) ≤ *
ECG Interpretation Rhythm Rate Axis P, QRS,Intervals ST & T changes Other abnormalities
Normal Sinus Rythm Positive P in I,II HR= Regular RR intervals PR>120msec
Heart Rates= 300/RR interval(large squares) or 1500/RR interval(small squares) Bazzet`s formula : QTc = QT/ √RR(sec)
Axis (mean QRS axis): normally -30 to +90 LAD: LVH LAFB Inf. MI RAD: Normal RVH Lat. MI LPFB
Normal ECG
P-wave Normal values 1. Polarity. up in all leads except aVR. 2. Duration. < 2.5 mm. 3. Amplitude. < 2.5 mm. Abnormalities 1. Inverted P-wave Junctional rhythm. 2. Wide P-wave (P- mitrale) LAE 3. Peaked P-wave (P-pulmonale) RAE 4. Saw-tooth appearance Atrial flutter 5. Absent P wave Atrial fibrillation
QRS complex Normal values Duration: < 2.5 mm. Morphology: progression from Short R and deep S (rS) in V1 to tall R and short S in V6 with small Q in V5-6 (qRs). Abnormalities : 1. Wide QRS complex Bundle branch block. Ventricular rhythm. 2. Tall R in V1 RVH. RBBB. Posterior MI. WPW syndrome. 3. abnormal Q wave [ > 25% of R wave] MI. Hypertrophic cardiomyopathy. Normal variant.
ST- segment Normally it's isoelectric. [i.e. at same level of RP segment] Abnormalities: 1. ST elevation: Acute MI. Prinzmetal angina. Acute pericarditis. Early repolarization. 2. ST depression: Ischemia. Ventricular strain. BBB. Hypokalemia. Digoxin effect.
T-wave Normal values. 1. Polarity: Always up in I,II,V4-5 Always down in aVR. Variable in III, aVL, aVF, V amplitude: < 10mm in the chest leads. Abnormalities: 1. Peaked T-wave: Hyper-acute MI. Hyperkalemia. Normal variant. 2. T- inversion: Ischemia. Myocardial infarction. Myocarditis Ventricular strain BBB. Hypokalemia. Digoxin effect.
QT- interval Definition: Time interval between beginning of QRS complex to the end of T wave. Normally: At normal HR: QT ≤ 11mm (0.44 sec) (or ) QTc = QT/ √RR Abnormalities: 1. Prolonged QT interval: hypocalcemia and congenital long QT syndrome. 2. Short QT interval: hypercalcemia.
Mitral P Pulmonar P
Criteria of ventricular enlargement LVH : 1. SV1 + (RV5 or RV6) ≥ 35 mm (or) RV5 or RV6 ≥ 25 mm 2. LV strain 3. LAE RVH : 1. Relatively tall R in V1 2. RV strain 3. RAD
LVH
RVH with RAE
Bundle system and sites of block
Complete Left Bundle Branch Block QRS duration ≥ 120 msec Broad, notched, or slurred R waves in leads I, aVL, V 5 and V 6 Small or absent initial r waves in right precordial leads (V 1 and V 2 ) followed by deep S waves Absent septal q waves in leads I, V 5, and V 6 Prolonged time to peak R wave (>60 msec) in V 5 and V 6
Complete Right Bundle Branch Block QRS duration ≥ 120 msec rsr ′, rsR ′,, or rSR ′, patterns in leads V 1 and V 2 S waves in leads I and V 6 ≥ 40 msec wide Normal time to peak R wave in leads V 5 and V 6 but >50 msec in V 1
Left Bundle branch block (LBBB)
Right Bundle branch block (RBBB)
Incomplete RBBB
Left anterior fascicular block (LAFB)
Bifascicular block (RBBB+LAFB)
Trifascicular block 1
ST segment and T wave changes in ischemia and MI
Lateral ischemia
Inferolateral ischemia
SVT with ischemia
NSTEMI
Evolution of ECG changes in MI
Hyperacute MI
Acute anteroseptal MI (STEMI)
Acute anterolateral MI (with hyperacute T)
Acute anterolateral MI
Acute inferoposterior MI
Right ventricular infarction
SAH
Hyperkalemia
Sever hyperkalemia
Atrial ectopic
Atrial bigeminy
Junctional ectopic
Ventricular ectopic with compensatory pause
Ventricular ectopic without compensatory pause
V. bigeminy
V. trigeminy
R on T event
R on T phenomena
Multifocal V. ectopics
V. Couplet
Tachycardia Wide or Narrow Regularity P wave P and QRS Rate and Association Axis Abnormalities
Sinus tachycardia
Sinus arrhythmia
Paroxysmal supraventricular tachycardia [PSVT]
SVT
SVT with retrograde P- wave
Atrial fibrillation [fine]
Atrial fibrillation [coarse]
AF + LBBB
Atrial flutter
Multifocal atrial tachycardia
Accelerated junctional rhythm
SUPPORTS SVTSUPPORTS VT Slowing or termination by vagal toneFusion beats Onset with premature P waveCapture beats RP interval ≤100 msecAV dissociation P and QRS rate and rhythm linked to suggest that ventricular activation depends on atrial discharge, e.g., 2 : 1 AV block rSR ′ V 1 P and QRS rate and rhythm linked to suggest that atrial activation depends on ventricular discharge, e.g., 2 : 1 VA block Long-short cycle sequence “Compensatory” pause Left-axis deviation; QRS duration >140 msec Specific QRS contours (see text) Major Features in the Differential Diagnosis of Wide QRS Beats Versus Tachycardia
Non-sustained VT
Ventricular tachycardia (VT)
VT
VT (with RBBB pattern)
VT (with LBBB pattern)
VT with capture beat
VT with fusion beat
Twisting VT (Torsades de pointes)
Ventricular fibrillation
Sinus bradycardia
Sinus arrhythmia
Junctional rhythm
Sinus arrest
Sino-atrial exit block
SA exit block
Sick sinus syndrome
First degree heart block
Second degree heart block Mobitz type I (Wenckebach block)
Second degree heart block Mobitz type II
Complete heart block
High degree AV block
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