Birth Trauma ( Birth Injuries)

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Presentation transcript:

Birth Trauma ( Birth Injuries) avoidable and unavoidable mechanical, hypoxic and ischemic injury affecting the infant during labor and delivery. Birth injuries may result from : Inappropriate or deficient medical skill or attention. They may occur, despite skilled and competent obstetric care The incidence:2-7/ 1000 live births.

Predisposing factors: Macrosomia, Prematurity, Cephalopelvic disproportion, Dystocia, Prolonged labor, and Breech presentation.

1. Cranial Injuries Erythema, abrasions, echymosis of facial or scalp tissues may be seen after forceps or vacuum assisted deliveries Subconjunctival ,retinal hemorrhages and petechiae of the skin of the head and neck: All are common. All are probably secondary to a sudden increase in intrathoracic pressure during passage of the chest through the birth canal. Parents should be assured that they are temporary and the result of normal hazards of delivery. Caput succedaneum: diffuse, sometimes echymotic edematous swelling of scalp soft tissue involving the part presenting during vertex presentation

It may extend across the midline and across suture lines. The edema disappears within the first few days of life. Analogous swelling, discoloration, and distortion of the face are seen in face presentations. No specific treatment is needed, but if there are extensive ecchymoses, phototherapy for hyperbilirubinemia may be indicated. Cephalhaematoma It is a subperiosteal haematoma most commonly lies over one parietal bone. It may result from difficult vacuum or forceps extraction

is differentiated from cephalohematoma by: Pulsation, An underlying skull fracture, usually linear and not depressed, is occasionally associated with cephalohematoma. require no treatment, although phototherapy may be necessary to ameliorate hyperbilirubinemia Incision and drainage are contraindicated because of the risk of introducing infection in a benign condition. A massive cephalohematoma may rarely result in blood loss severe enough to require transfusion. Cranial meningocele is differentiated from cephalohematoma by: Pulsation, Increased pressure on crying, and the Radiologic evidence of bony defect. Most cephalohematomas are resorbed within 2 wk-3 mo, depending on their size. They may begin to calcify by the end of the 2nd wk.

Cephalhematoma Caput succedaneum

Fractures of Skull Forceps or from The maternal symphysis pubis. May occur as a result of pressure from : Forceps or from The maternal symphysis pubis. Sacral promontory, or Ischial spines. a. Linear fractures, the most common, cause no symptoms and require no treatment. b. Depressed fractures are usually indentations similar to a dent in a Ping-Pong ball; they usually are a complication of forceps delivery or fetal compression.

Affected infants may be asymptomatic unless there is associated intracranial injury. It is advisable to elevate severe depressions to prevent cortical injury from sustained pressure.

Intracranial hemorrhages Causes: Sudden compression and decompression of the head as in breech and precipitate labour. Marked compression by forceps or in cephalopelvic disproportion. Fracture skull Predisposing factors: Prematurity due to physiological hypoprothrombinaemia, fragile blood vessels and liability to trauma. Asphyxia due to anoxia of the vascular wall . Blood diseases.

Types: Subdural Subarachnoid IVH Intracerebral Clinical picture: 1- Altered consciousness. 2- Flaccidity. 3- Breathing is absent, irregular and periodic or gasping. 4- Eyes: no movement, pupils may be fixed and dilated. 5- Opisthotonus, rigidity, twitches and convulsions. 6- Vomiting . 7- High pitched cry.    8- Anterior fontanelle is tense and bulging. 9- Lumbar puncture reveals bloody C.S.F.

Investigations: Ultrasound is of value. CT scan is the most reliable. MRI Treatment: Supportive care Treatment of seizures, anemia, shock, acidosis LP is diagnostic and therapeutic to relieve the intracranial tension Vit K, FFP, Antibiotics Symptomatic subdural hemorrhage in large term infants should be treated by removing the subdural fluid collection by means of a spinal needle placed through the lateral margin of the anterior fontanel.

Peripheral Nerves Injuries 1. Erb s palsy: C5,C6 ( C7 in 50% of cases), occur in less than 0.5% of deliveries , often associated with shoulder dystocia and breech or forceps deliveries. Arm held limply adducted, internally rotated, and pronated with wrist flexed and fingers flexed (“waiter’s tip” position)

2. Klumpke paralysis? A brachial plexus palsy involving injury to the lower plexus (C8, T1). It is associated with weakness of the flexor muscles of the wrist and the small muscles of the hand (“claw hand”). Treatment: In upper arm paralysis, the arm should be abducted 90 degrees with external rotation at the shoulder, full supination of the forearm, and slight extension at the wrist with the palm turned toward the face. This position may be achieved with a brace or splint during the 1st 1-2 wk. Immobilization should be intermittent throughout the day while the infant is asleep and between feedings.

In lower arm or hand paralysis, the wrist should be splinted in a neutral position, and padding placed in the fist Gentle massage and range-of-motion exercises may be started by 7-10 days of age. If the paralysis persists without improvement for 3 months, neuroplasty, neurolysis, end-to-end anastomosis, and nerve grafting offer hope for partial recovery. Botulism toxin may be used to treat biceps-triceps co-contractions. 3. Facial N palsy: It is usually due to pressure by the forceps blade on the facial nerve Spontaneous recovery usually occurs within 14 days.

Liver, spleen and kidney) Visceral Injuries: Liver, spleen and kidney) may be injured in breech delivery which should be avoided by holding the fetus from its hips. Hepatic rupture may result in the formation of a subcapsular hematoma. The hematoma may be large enough to cause anemia. Shock and death may occur if the hematoma breaks through the capsule into the peritoneal cavity.

A mass may be palpable in the right upper quadrant; the abdomen may appear blue. Early suspicion by means of ultrasonographic diagnosis and prompt supportive therapy can decrease the mortality of this disorder. Surgical repair of a laceration may be required. Rupture of the Spleen: May occur alone or in association with rupture of the liver. The causes, complications, treatment, and prevention are similar.

Adrenal Hemorrhage: Occurs with some frequency, especially after breech delivery in LGA infants or infants of diabetic mothers. 90% are unilateral; 75% are right sided. The symptoms are profound shock and cyanosis If suspected, abdominal ultrasonography may be helpful, and treatment for acute adrenal failure may be indicated

Skeletal Injuries The clavicle: more frequently fractured than any other bone; Treatment, consists of immobilization of the arm and shoulder on the affected side. Humerus, Femur: The prognosis is excellent for fractures of the extremities. Sternomastoid injury: Exaggerated lateral flexion of the neck leading to torticollis and swelling in the muscle. It is usually improved within 2 weeks but permanent torticollis may continue.

Necrotizing Enterocolitis( NEC) Necrotizing enterocolitis (NEC) is the most common gastrointestinal (GI) medical/surgical emergency occurring in neonates. the condition is characterized by variable damage to the intestinal tract ranging from mucosal injury to full-thickness necrosis and perforation. NEC affects close to 10% of infants who weigh less than 1500 g, with mortality rates of 50% or more depending on severity. Although it is more common in premature infants, it can also be observed in term and near-term babies.

Although various clinical and radiographic signs and symptoms are used to make the diagnosis, the classic clinical triad consists of abdominal distension, bloody stools, and pneumatosis intestinalis. Occasionally, signs and symptoms include temperature instability, lethargy, or other nonspecific findings of sepsis. The distal part of the ileum and the proximal segment of the colon are involved most frequently THE TRIAD OF INTESTINAL ISCHEMIA ( INJURY),ENTERAL NUTRITION, AND PATHOGENIC ORGANISMS HAD CLASSICLY BEEN LINKED TO NEC

The major risk factor for NEC is prematurity NEC probably result from an interaction between loss of mucosal integrity due to : ischemia, infection, inflammation , and the hosts response to that injury( circulatory, immunologic ,inflammatory) resulting in necrosis of the affected area coagulation necrosis is the histological finding of intestinal specimens Various bacterial and viral agents( E coli, Klebsiella, cl. Perfringens , staph epidermidis and rota virus have been recovered from cultures , nonetheless; no pathogen is identified in most cases

Aggressive enteral feeding may predispose to the development of NEC Signs & Symptoms: Systemic GI Lethargy Abdominal distension Apnea/ respiratory distress Abdominal tenderness Temperature instability Feeding Intolerance (Not right) Delayed gastric emptying Acidosis Vomiting Glucose instability Occult/ gross blood in stool Poor perfusion/ shock Change of stool pattern/diarrhea DIC Abdominal mass Positive results of blood culture Erythema of the abdominal wall

Diagnosis: High index of suspicion Plain abdominal x-ray: pneumatosis intestinalis, portal vein gass, pneumoperitoneum Hepatic US: portal venous gas despite normal abd x ray DDX: Specific infections, GI obstruction, volvulus, isolated intestinal perforation Treatment: Supportive care Cessation of feeding, nasogastric decompression, IV fluids

3. Careful attention to respiratory status, coagulation profile, acid-base and electrolyte balance 4. Blood culture and antibiotics that covers gram negative, positive and anaerobic 5. Removal of umbilical catheter6. close monitoring: physical assessment, serial abdominal x- rays 6. Indications of surgery: a. evidence of perforation b. positive abdominal paracentesis Failure of medical treatment, a single fixed bowel loop on x ray, abdominal wall erythema, palpable mass are relative indications for surgery

Prevention: Breast feeding Minimal enteral feeding probiotics