Sleep EEG patterns in infants with congenital Zika virus syndrome

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Presentation transcript:

Sleep EEG patterns in infants with congenital Zika virus syndrome Maria Durce Costa Gomes Carvalho, Demócrito de Barros Miranda-Filho, Vanessa van der Linden, Paula Fabiana Sobral, Regina Coeli Ferreira Ramos, Maria Ângela Wanderley Rocha, Marli Tenório Cordeiro, Sarah Pinheiro de Alencar, Magda Lahorgue Nunes Clinical Neurophysiology Volume 128, Issue 1, Pages 204-214 (January 2017) DOI: 10.1016/j.clinph.2016.11.004 Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 1 (A–C) Modified hypsarrhythmic patterns recorded in patients 26, 29 and 32. Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 1 (A–C) Modified hypsarrhythmic patterns recorded in patients 26, 29 and 32. Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 2 (A–H) Evolution of an electrographic seizure without clinical manifestation. High voltage (200uV) slow waves (2Hz) were recorded in the left occipital region (A, B), as voltage increases the discharges turned more rhythmic (C, D) and latter spreading to the right hemisphere (E–G), by the end of the episode the activity is again restricted to left occipital region (H). (Patient 11). Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 2 (A–H) Evolution of an electrographic seizure without clinical manifestation. High voltage (200uV) slow waves (2Hz) were recorded in the left occipital region (A, B), as voltage increases the discharges turned more rhythmic (C, D) and latter spreading to the right hemisphere (E–G), by the end of the episode the activity is again restricted to left occipital region (H). (Patient 11). Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 2 (A–H) Evolution of an electrographic seizure without clinical manifestation. High voltage (200uV) slow waves (2Hz) were recorded in the left occipital region (A, B), as voltage increases the discharges turned more rhythmic (C, D) and latter spreading to the right hemisphere (E–G), by the end of the episode the activity is again restricted to left occipital region (H). (Patient 11). Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 2 (A–H) Evolution of an electrographic seizure without clinical manifestation. High voltage (200uV) slow waves (2Hz) were recorded in the left occipital region (A, B), as voltage increases the discharges turned more rhythmic (C, D) and latter spreading to the right hemisphere (E–G), by the end of the episode the activity is again restricted to left occipital region (H). (Patient 11). Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions

Fig. 3 (A and B) (A) Focal, bilateral frontal, slow (0.5Hz) waves (Patient 19) and (B) Focal frontal slow waves and intermixed spikes (Patient 18). Clinical Neurophysiology 2017 128, 204-214DOI: (10.1016/j.clinph.2016.11.004) Copyright © 2016 International Federation of Clinical Neurophysiology Terms and Conditions