Figure 1 Food, nutrition, obesity, physical activity, and the cellular processes linked to cancer Figure 1 | Food, nutrition, obesity, physical activity,

Slides:



Advertisements
Similar presentations
Figure 1 The heterogenous landscape of triple-negative breast cancer
Advertisements

Reproduced with permission from Cronin M et al
Figure 1 Estimated annual percentages of new
Figure 1 CTLA-4 and PD-1–PD-L1 immune checkpoints
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 5 Mutational heterogeneity in oesophageal and gastric cancer
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 2 Multiscale modelling in oncology
Figure 1 Cellular processes involved in cancer development
Figure 5 Schematic illustration of different clinical trial designs
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 1 Immunomodulatory monoclonal antibodies and armoured chimeric antigen receptor (CAR) T cells overcome immune suppression Figure 1 | Immunomodulatory.
Figure 2 Signalling pathways and physiological domains that are
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 3 Monitoring clonal evolution using liquid biopsies
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 2 Copy-number variations in multiple myeloma
Figure 5 Identification of mucinous carcinoma
Figure 1 Underreporting of treatment-related toxicities by physicians, relative to patients with either advanced-stage lung cancer, or early-stage breast.
Figure 2 Therapeutic targeting of the PI3K/AKT/mTOR pathway
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 1 Proposed treatment algorithm for advanced gastroesophageal cancer based on publish recommendations Figure 1 | Proposed treatment algorithm for.
Figure 1 CAR-T-cell design
Figure 1 A schematic representation of the HER2 signalling pathway
Figure 4 Effects of delaying cardioprotective medications after anthracycline administration Figure 4 | Effects of delaying cardioprotective medications.
Figure 1 Cost of one month of treatment with
Figure 2 The association between CD8+ T‑cell density of the tumour
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 3 Drug cycling with collateral sensitivity
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 2 Differences between MC and AC
Figure 3 Possible modalities for reconciliation of patient's and physician's report of symptomatic treatment-associated toxicities Figure 3 | Possible.
Figure 1 A schematic representation of the role
Figure 1 Balance between proteasomal load and capacity
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 1 NIR fluorescence is more suitable for
Figure 2 Metabolic heterogeneity in tumours
Figure 4 Treatment plans using stereotactic body radiotherapy (SBRT)
Figure 3 Clinical trial design in charged-particle therapy (CPT)
Figure 3 The yin and yang of tumour-associated
Figure 2 Median monthly launch price of a new anticancer drug,
Figure 1 Translocations involved in multiple myeloma
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 2 Key features of gastric cancer subtypes according to The Cancer Genome Atlas (TCGA) Figure 2 | Key features of gastric cancer subtypes according.
Figure 2 Host immune responses, not the radiosensitivity
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 2 Effects of imatinib on components of the anticancer immunosurveillance system Figure 2 | Effects of imatinib on components of the anticancer immunosurveillance.
Figure 5 Palliative-care-referral patterns
Figure 2 Frequency and overlap of alterations
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 3 Tumours secrete factors that cause systemic immunosuppression
Figure 1 Mechanisms of action of immunotherapy modalities
Figure 2 Effect of chromosomal instability tolerance
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 4 Radiogenomics analysis can reveal relationships
Figure 2 Radiomics in cardiology
Figure 1 Overview of the imaging biomarker roadmap
Figure 3 Determination of the primary site
Figure 2 Signalling downstream of the IL-6 receptor
The impact of FIH-1 on ASPP2 protein interactions.
Figure 1 Gene-expression quantification methods
Figure 2 Mechanisms of RET activation in cancer
Presentation transcript:

Figure 1 Food, nutrition, obesity, physical activity, and the cellular processes linked to cancer Figure 1 | Food, nutrition, obesity, physical activity, and the cellular processes linked to cancer6. Nutritional factors can influence cancer development by affecting fundamental cellular processes, including those that regulate the balance between cell proliferation and death, appropriate cell differentiation, the expression of oncogenes and tumour-suppressor genes, cell signalling, and other factors in the cellular environment (such as hormonal factors) that influence gene expression. Thus, bioactive food components can interact with gene expression at multiple points, and furthermore, a specific food typically contains numerous components that interact additively or even synergistically with these cellular processes. Permission obtained from World Cancer Research Fund International © World Cancer Research Fund/American Institute for Cancer Research Food, Nutrition, Physical Activity, and the Prevention of Cancer: a Global Perspective (AICR, Washington District of Columbia, USA, 2007). Permission obtained from World Cancer Research Fund International © World Cancer Research Fund/American Institute for Cancer Research Food, Nutrition, Physical Activity, and the Prevention of Cancer: a Global Perspective (AICR, Washington District of Columbia, USA, 2007) Mayne, S. T. et al. (2006) Diet, nutrition, and cancer: past, present and future Nat. Rev. Clin. Oncol. doi:10.1038/nrclinonc.2016.24