Fig. 3. Expression of Ninj1protein in cortices of the ischemic hemispheres of the rat brain at 4 day post-MCAO. Coronal brain sections were prepared at.

Slides:



Advertisements
Similar presentations
Fig. 2. The expression of pKr-2 in the substantia nigra (SN) of patients with Parkinson's disease (PD). (A, B) Western blot analysis for pKr-1-2 and pKr-2,
Advertisements

Volume 9, Issue 2, Pages (February 2004)
FIGURE 2. The regional cerebral blood flow decreased approximately 85% below the baseline within 30 seconds of the insertion of the occlusion suture. The.
Fig. 1. In situ hybridization analysis for detecting endogenous mBest1 transcript in whole-brain region. In situ hybridization results from coronal (top.
Fig. 2. Post-treatment with isoflurane reduced infarct volume
Α-Lipoic Acid Promotes Neurological Recovery After Ischemic Stroke by Activating the Nrf2/HO-1 Pathway to Attenuate Oxidative Damage Cell Physiol Biochem.
Cerebrovasc Dis 2016;42: DOI: /
Fig. 1. Infarct size and behavioral outcomes in response to single and repeated treatment of hUCB-MSCs after MCAO. (A) Schematic of the time schedule.
Fig. 2. Infection by lentivirus
From: Blocking Endothelin-B Receptors Rescues Retinal Ganglion Cells from Optic Nerve Injury through Suppression of Neuroinflammation Invest. Ophthalmol.
From: Defective Angiogenesis and Intraretinal Bleeding in Mouse Models With Disrupted Inner Retinal Lamination Invest. Ophthalmol. Vis. Sci ;57(4):
Fig. 2. Expression of Ninj1 in cortices of ischemic hemispheres of rat brains at 1 day post-MCAO. Coronal brain sections were prepared at 1 day post-MCAO.
A B C D % positive cells 10 % Apoptotic nuclei 10 Figure S5 PM
Fig. 4. Reactive cerebral artery extraction using standard deviation (SD) map categorization is shown. (A) Image processing steps for the reactive artery.
Cell Physiol Biochem 2016;40: DOI: /
Youngshik Choe, Julie A. Siegenthaler, Samuel J. Pleasure  Neuron 
Prevention of seizures and reorganization of hippocampal functions by transplantation of bone marrow cells in the acute phase of experimental epilepsy 
Increased astrocyte proliferation is paralleled with reduced infiltration of immune cells in mice with inducible SMOM2 expression in astrocytes at 5 dpi.
The Spatial and Temporal Expression Patterns of Integrin α9β1 and One of Its Ligands, the EIIIA Segment of Fibronectin, in Cutaneous Wound Healing  Purva.
Intra-articular injection of human mesenchymal stem cells (MSCs) promote rat meniscal regeneration by being activated to express Indian hedgehog that.
ApoE4 Accelerates Early Seeding of Amyloid Pathology
Volume 10, Issue 19, Pages (October 2000)
Engineered Exosomes as Vehicles for Biologically Active Proteins
Johanna Sigl-Glöckner, Michael Brecht  Cell Reports 
Volume 26, Issue 1, Pages (January 2018)
Anti-tumor effect of anti-PD-1 antibody against SCC3 tumor
Fig. 7 CSPG4-high GBMs show more microglia than CSPG4-low GBMs and express TNFα. CSPG4-high GBMs show more microglia than CSPG4-low GBMs and express TNFα.
BDNF expression in the cerebellum and brain stem region.
Fig. 3. Morphological changes associated with glial activation were reduced in 16-month-old APP/PS1;C3 KO mice. Morphological changes associated with glial.
Volume 17, Issue 5, Pages (May 2009)
Volume 19, Issue 3, Pages (April 2017)
Neuroprotection from Stroke in the Absence of MHCI or PirB
DFP-induced astrogliosis is reduced by urethane.
Volume 13, Issue 3, Pages (March 2006)
Distribution of splenic T cells, B cells, and NK cells in NSG hL-7xhIL-15 humanized mice. Distribution of splenic T cells, B cells, and NK cells in NSG.
Fig. 1. Overview of the nervous system of the adult S. roscoffensis.
Temporal dynamics of the primitive streak and mesoderm progenitors in β-catenin-activated hES cells. Temporal dynamics of the primitive streak and mesoderm.
Robo3 in the GnRH neuronal system.
Volume 26, Issue 1, Pages (January 2018)
Fig. 8. The morphology of the ventral nerve cord in Ror-Myc overexpressing embryos is normal. The morphology of the ventral nerve cord in Ror-Myc overexpressing.
Fig. 2. Immunostaining for ClC-5 in isolated hog gastric mucosa
Fig. 2. Deficiency of neuronal HS leads to reduced neuroinflammation.
Activation of glial cells.
Fig. 4. Reduction of apoptosis and autophagic stress by treatment with silibinin in the KA-treated hippocampus. (A, B) Double-immunofluorescence staining.
Fig. 3. Neuroprotective effects of silibinin against KA-induced excitotoxicity. (A) Silibinin was intraperitoneally injected for 8 days, starting 1 day.
Fig. 5. Anti-inflammatory effects of silibinin on KA treatment in the hippocampus. (A) Representative coronal sections of the hippocampal CA1 region following.
Neuronal and glial expression of Nogo-A and synaptic localization of Nogo-A and NgR 1 in the motor cortex. Neuronal and glial expression of Nogo-A and.
Volume 16, Issue 2, Pages (February 1996)
Volume 21, Issue 1, Pages (January 2013)
PTZ-induced neuronal activity visualized by IEGs
Fig. 2. Infarct size and mNSS, weight change of animals with oral gavage of HSA and saline. (A) TTC staining of the individual brain was shown that the.
Fig. 3. Morphological changes associated with glial activation were reduced in 16-month-old APP/PS1;C3 KO mice. Morphological changes associated with glial.
TOMA does not affect NFTs in posterior hypothalamus or cortex.
Fig. 3. GABA content was increased in reactive astrocytes from 3D culture and hippocampal brain slice. (A) Immunostaining of neuron-astrocyte 3D co-culture.
At 1 month, control and les rats were fed AL or fasted every other day for IF. Representative images of 1 μm sections, with electron micrograph insets.
Brain ischemic tolerance model using mouse MCAO
Absence of PC-induced ischemic tolerance in P2X7−/− mice.
Fig. 2. TH expression is decreased in mice with HFD-induced obesity
Fig. 2 IRF8 is expressed in CD68+ macrophages after SCI.
Fig. 6. Microglial phagocytosis and lysosomal uptake of Aβ induced by SUS treatment. Microglial phagocytosis and lysosomal uptake of Aβ induced by SUS.
Fig. 3. HERV-K–induced neuronal toxicity in vivo.
Fig. 3. LPS induces degeneration of DA neurons and microglial activation in the SN in vivo. LPS (5 µg/3 µl) was unilaterally injected into the SN in the.
Fig. 9. Scriptaid and RGFP966 have no effect on Iba-+1 and CD4+ cell immune cell infiltration after SCI. BALB/c mice were subjected to a T-cut hemisection.
Fig. 3. hALDH1L1 promoter-driven Cre recombinase-mediated tdTomato expression in BLA. (A) Low magnification images of injection area in amygdala. Each.
Passive immunization with CT α-syn antibodies improved TH pathology and neuroinflammation in the striatum of α-syn tg mice. Passive immunization with CT.
Expression of Rapgef2 and Rapgef6 in the E13.5 and E15.5 brains.
rat EDL mouse gastrocnemius S1 Fig. Isotype control.
Fig. 1. The brain of Octopus minor. (A) Left: a live specimen of O
Fig. 5. Phagocytosis of zymosan particles by BV2 cells
Volume 9, Issue 2, Pages (February 2004)
Presentation transcript:

Fig. 3. Expression of Ninj1protein in cortices of the ischemic hemispheres of the rat brain at 4 day post-MCAO. Coronal brain sections were prepared at 4 days post-MCAO and immunostained with anti-Ninj1 antibody (A, G) or anti-Ninj1- and anti-CD45 (B, H), anti-Iba-1 (C, I), anti-anti-RECA (D), anti-NeuN (E), or anti-GFAP (F) and DAPI. The pictures shown are representative of three independent experiments. The insets in A-D, G-I are high magnification photographs of each image. Scale bars in A and G represent 1 mm, those in B-F and H-I represent 20 µm, and those in insets represent 10 µm. Fig. 3. Expression of Ninj1protein in cortices of the ischemic hemispheres of the rat brain at 4 day post-MCAO. Coronal brain sections were prepared at 4 days post-MCAO and immunostained with anti-Ninj1 antibody (A, G) or anti-Ninj1- and anti-CD45 (B, H), anti-Iba-1 (C, I), anti-anti-RECA (D), anti-NeuN (E), or anti-GFAP (F) and DAPI. The pictures shown are representative. . . Exp Neurobiol. 2016 Apr;25(2):64-74. http://dx.doi.org/10.5607/en.2016.25.2.64