Determinants of Eating Behaviour in European Children, Adolescents and their Parents Overview & Key Findings Wolfgang Ahrens (I.Family coordinator) University.

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Presentation transcript:

Determinants of Eating Behaviour in European Children, Adolescents and their Parents Overview & Key Findings Wolfgang Ahrens (I.Family coordinator) University of Bremen & Leibniz Institute for Prevention Research & Epidemiology – BIPS Deputy coordinators: Alfonso Siani & Iris Pigeot

Aim: contribute to reducing burden of nutrition-related diseases Understand interplay between barriers and main drivers of a healthy food choice Identify predictors of unnecessary weight gain and cardio-metabolic risk by linking them to diet, physical activity and interacting factors Focus on child and his / her family Assess how different factors affect children as they grow up Develop and convey strategies to induce changes towards a healthy behaviour

Partners Strovolos, Cyprus Ghent, Belgium Copenhagen, Denmark Tallin, Estonia Helsinki, Finland Bremen, Germany Pécs, Hungary Avellino, Italy Milan, Italy Utrecht, Netherlands Palma de Mallorca, Spain Zaragoza, Spain Gothenburg, Sweden Bristol, United Kingdom Lancaster, United Kingdom Andover, United Kingdom

Community intervention Timeline of recruitment and follow-up IDEFICS – I.Family cohort, starting with 2-10 year olds 2007 2008 2009 2010 2011 2012 2013 2014 2015 2016 2017 2006 Community intervention Baseline 16,228 children Follow-up 1 13,596 children Follow-up 2 9,617 children Contrasting groups Transition: toddler  child Transition: child  adolescent Transition: adolescent  adult

Questionnaires & examinations 1 Questionnaires (parent + child) Social factors, lifestyle, peers + physical activity Food frequency, preference, eating behaviour Medical history Family members + household 24-hour dietary recall Web-based dietary recall Physical activity Accelerometer: 7 days Built environment: GIS + GPS

Questionnaires & examinations 2 Physical examinations Anthropometry + blood pressure Bone health: ultrasound Biological markers Blood, saliva + urine Specific tests in subgroups Sensory taste perception Neuropsychological tests: impulsivity Brain mechanisms of food choice: fMRI

Prevalence of childhood overweight/obesity (2-10 yr)

Children with low socio-economic background Dietary behaviour Children with low socio-economic background Persistently unhealthier dietary profiles over a 2-year period Dietary patterns rich in fruits & vegetables, wholemeal cereals, and low in animal products Lower risk of overweight/obesity Less 2-year weight gain

Sleep and weight status Short sleep duration  being overweight – particularly in primary school children Inverse relationship between sleep duration and BMI  mainly explained by inverse association between sleep duration & fat mass Insulin may explain part of this association, in particular in heavier children

Physical activity (PA) and overweight (OW) Few children meet physical activity guidelines (60min MVPA/day) Causality goes both ways Higher or increasing fat mass  decline in MVPA Just 10 minutes more MVPA per day  prevent excess weight gain MVPA = Moderate to Vigorous Physical Activity

Built environment and physical activity Physical activity-friendliness of the built environment (“moveability”)  more MVPA of 596 primary school children in the German study region Playground density and density of playgrounds and parks combined  positive effects on MVPA MVPA = Moderate to Vigorous Physical Activity

Media consumption TV exposure  preference for sugary/fatty foods  followed by higher consumption of sugar-sweetened beverages  increased risk of overweight/obesity One-third of children exceeded screen time recommendations (max. 2h/day) Exceeding sedentary guidelines  increased risk of high blood pressure Watching TV during meals, having a TV in the child’s bedroom and watching TV more than 1h/day  being overweight/obese

Swimming upstream The causes of obesity – and the causes of the causes Adapted from: Swinburn et al. The global obesity pandemic: shaped by global drivers and local environments. Lancet. 2011; 378: 804-14

“Childhood obesity undermines the physical, social and psychological wellbeing of children and is a known risk factor for adult obesity and non-communicable diseases. There is an urgent need to act now to improve the health of this generation and the next.” WHO 2016

Ending Childhood Obesity – strategic objectives No single intervention can halt the rise of the growing obesity epidemic. To successfully challenge childhood obesity requires addressing the obesogenic environment as well as critical elements in the life-course.

Thank you!