Saskia Z. H. Rittersma, Martijn Meuwissen, Chris M

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Eosinophilic infiltration in restenotic tissue following coronary stent implantation Saskia Z.H. Rittersma, Martijn Meuwissen, Chris M. van der Loos, Karel T. Koch, Robbert J. de Winter, Jan J. Piek, Allard C. van der Wal Atherosclerosis Volume 184, Issue 1, Pages 157-162 (January 2006) DOI: 10.1016/j.atherosclerosis.2005.03.049 Copyright © 2005 Elsevier Ireland Ltd Terms and Conditions

Fig. 1 Part of an atherectomy specimen derived from an in-stent restenotic lesion A: macroscopic view of the retrieved atherectomy specimen, showing partially removed stent struts embedded in bluish-white restenosis tissue (white arrows). B: anti-CD3 stained section, showing clusters of T cells around the empty spaces corresponding with the sites of stent struts. Boxed area in detail shown in C. D: anti-CD40L staining, showing a subpopulation of activated T cells within the infiltrate. E: anti-eosinophil staining. Atherosclerosis 2006 184, 157-162DOI: (10.1016/j.atherosclerosis.2005.03.049) Copyright © 2005 Elsevier Ireland Ltd Terms and Conditions

Fig. 2 Sections of atherectomy specimen derived from a restenotic lesion after balloon angioplasty. Yellow pigment (arrows) represents ceroid pigment (endpoint of lipid peroxidation). A: overview, anti-CD68 stained section, showing extensive areas of closely packed macrophages B: detailed section of 3a, indicating massive infiltration of macrophages in lipid rich tissue. C: detailed anti-CD3 stained section, showing T cells associated with ceroid pigment D: anti-CD3/CD25 stained section illustrating a few interleukin-2 receptor expressing T cells as a marker of recent onset activation. Atherosclerosis 2006 184, 157-162DOI: (10.1016/j.atherosclerosis.2005.03.049) Copyright © 2005 Elsevier Ireland Ltd Terms and Conditions