The lymphocyte markers that distinguish the various stages of B-cell differentiation are shown. Markers that are expressed on the cell surface are indicated.

Slides:



Advertisements
Similar presentations
Metabolism of ganglioside GM2 in GM2 activator-deficient fibroblasts
Advertisements

A: Population frequency histogram of urinary debrisoquine/4-hydroxydebrisoquine ratios after debrisoquine administration to 258 individuals in a healthy.
Genomic organization of the human cathepsin K gene
More detailed view of the process shown in Fig
Osmotic gradient ektacytometry of red blood cells with varying degrees of spectrin deficiency. In the spectrin-deficient cells, the minimum deformability.
A, Diagnosis of clinical severity (mild, moderate, severe) of hemophilia is based on in vitro coagulant activity as shown. B, Distribution of severe and.
Spectrum of different types of human gene mutations logged in Human Gene Mutation Database as of September 13, 1998 ( Source: The.
Combinatorial interactions of cyclins and cyclin-dependent kinases (cdks) during the cell cycle. Progression from G0 through the restriction point in G1.
Advantages of isolated liver transplantation before kidney failure over combined liver-kidney transplantation afterwards. Oxalate excretion: Black line.
Percentile lines for the relationship between insulin sensitivity (SI) and the first-phase insulin response (AIRglucose) based on data from 93 normal subjects.45.
Reverse-dot ASO analysis for various point mutations causing cystic fibrosis. For each mutation, the PCR product is hybridized to an ASO dot on the left.
Morphologic appearance of EH skin. Shown is a semithin section (0
Distribution of diagnoses among the nearly 115,000 cases of lymphoid malignancy diagnosed in There are 5400 cases of acute lymphoid leukemia (ALL),
Distribution of diagnoses among the nearly 115,000 cases of lymphoid malignancy diagnosed in There are 5400 cases of acute lymphoid leukemia (ALL),
Metabolic pathways for the formation and disposal of adenine and adenosine in humans, compared with those for hypoxanthine. Adenine phosphoribosyltransferase.
IX; formation of sphingolipids, sphingosine is synthesised from L-serine and palmitoyl-CoA. Source: Serine deficiency disorders, The Online Metabolic and.
Proposed model for the structure of ABC1 within the plasma membrane (modified from reference202 ). The two symmetric halves consisting of six membrane-spanning.
Illustration of the origin of tubular cysts
Light micrographs comparing pycnodysostosis and normal bone
Schematic representation of the DTDST protein
Radiographic appearance of intracranial meningioma in NF2
The PKD1 and PKD2 gene products are integral transmembrane proteins
Agarose gel electrophoresis (A,B,C,D) and two-dimensional nondenaturing polyacrylamide gradient gel electrophoresis (E,F) of plasma from a normolipidemic.
Crystal structure of human short/branched chain acyl-CoA dehydrogenase (PDB file 2JIF). A homotetramer with bound butyryl-Co as substrate is represented.
A section of the cerebellum stained with Sevier-Munger preparation from the brain of a 10-year-old SCA1 patient with 82 CAG repeats who died after 6 years.
Effect of chenodeoxycholic acid feeding on the bilirubin levels of a 10-year-old boy with 3β-hydroxy-Δ5-oxidoreductase/isomerase deficiency. (From Setchell.
Aggregation-mediated passive sorting of secretory and membrane proteins during the biogenesis of a secretory granule in the TGN. (1) The luminal contents.
Urinary excretion (mg/h) of tryptophan derivatives following administration of oral L-tryptophan (about 70 mg per kilogram body weight) administered to.
Neuropathology. Photomicrographs of the cerebellum
Free energy folding landscape for a hypothetical protein
Representation of Southern blot analysis revealing an expanding triplet repeat mutation in a myotonic dystrophy family. A grandmother and mother with the.
Progressive myopathy in a patient with type IIIa glycogen storage disease. The patient has a debrancher deficiency in both liver and muscle (subtype IIIa).
Model depicting the cycling of REP
Basal and peak serum thyrotropin (TSH) concentrations following intravenous administration of 500 μg thyrotropin-releasing factor (TRF) to subjects with.
Relation between the fractional catabolic rate (FCR) for plasma LDL and the number of LDL receptors on fibroblasts in patients with FH. The values for.
Model accounting for the mechanisms linking epipodophyllotoxin therapy, MLL fusion proteins, cell cycle progression, and the relaxation of cell cycle checkpoints,
Two children with LPI. The pictures were taken at the time of diagnosis. A, Child 12 years old. B, Child 6 years old. Note the prominent abdomen, hypotrophic.
Evolution of the ubiquitous α-, β-, and γ-crystallins and their structure. Details are described in the text. The α-crystallins are related to heat-shock.
Depiction of meiotic crossing over and linkage analysis
Anteroposterior radiographs of right tibia and fibula of case 2 at 2 years of age and left tibia and fibula of case 3 at age 6 years. Features of osteopetrosis.
Localization of WT1 mRNA expression in the developing kidney
Mechanism of NO synthesis
A comparison of normal cardiac anatomy (A) and the pathology of hypertrophic cardiomyopathy (B). Hypertrophic cardiomyopathy causes cardiomegaly (570 g;
Structure and mutation spectrum of BRCA1
Orofacial dyskinesia in a patient with Huntington's disease
OCTN2 mRNA in fibroblasts from normal controls and patients with primary carnitine deficiency. PolyA RNA isolated from fibroblasts of normal controls or.
Mechanism of NO synthesis
Insulin secretion rates during graded intravenous glucose infusions administered to 6 MODY-2 subjects with glucokinase (GCK) mutations and 6 control subjects.
Photosensitive skin lesions on the face of a child with C4 deficiency
Expression of mutant human P5C dehydrogenase alleles in yeast
Karyotype of glioblastoma
Hydrolysis of membrane-associated ceramide by acid ceramidase in the presence of saposins and bis(monoacylglycero)phosphate (BMP) in the acidic lysosomal.
MRI contrast-enhancement and progression of X-ALD
Key structural elements of p53 DNA binding
Electrophoregram profiles showing the 35delG mutation of CX26 (GJB2) in an individual with recessively inherited hearing loss, and in his heterozygous.
Corneal opacity in a heterozygote observed by slit-lamp microscopy
FISH of glioblastoma. This interphase nucleus from a glioblastoma contains three chromosome 7 centromere signals (dark) and one centromere 10 signal (light).
A model of the protein C activation complex
Young female with OCA1B. The hair was white at birth
Overview of the TGF-β signaling pathway
The effects of apo E alleles on various lipoprotein parameters
Hepatic glucose production as a function of fasting plasma glucose levels. These studies involved the same individuals who were studied in Fig
Basic algorithm for cancer risk assessment that employs gene testing.
Schematic working model of the hepatic microsomal glucose-6-phosphatase system. ER = endoplasmic reticulum. This model is not meant to represent the actual.
Detection of XIST RNA in interphase cells from a normal male, a normal female, and a 49,XXXXX female by RNA fluorescence in situ hybridization. Male.
Structure and mutation spectrum of BRCA2
Composite megalosaccharide proposed for blood group substance
Complete chemical structures of the neutral glycosphingolipids that accumulate in Fabry disease. A, Globotriaosylceramide, the major accumulated substrate.
The PBDs can be caused by defects in peroxisomal matrix protein import or peroxisome membrane synthesis. Immunofluorescence microscopy shows that human.
DNA analysis on patients with STS deficiency
Presentation transcript:

The lymphocyte markers that distinguish the various stages of B-cell differentiation are shown. Markers that are expressed on the cell surface are indicated as solid lines; markers that are predominantly cytoplasmic are shown as dotted lines. Note that some markers may move from the cytoplasm to the cell surface. Source: Antibody Deficiencies, The Online Metabolic and Molecular Bases of Inherited Disease Citation: Valle D, Beaudet AL, Vogelstein B, Kinzler KW, Antonarakis SE, Ballabio A, Gibson K, Mitchell G. The Online Metabolic and Molecular Bases of Inherited Disease; 2014 Available at: http://ommbid.mhmedical.com/DownloadImage.aspx?image=/data/books/971/ch184fg3.png&sec=62652905&BookID=971&ChapterSecID=62652864&imagename= Accessed: October 18, 2017 Copyright © 2017 McGraw-Hill Education. All rights reserved

Feedback: Privacy Policy Feedback
Do Not Sell My Personal Information
About project: SlidePlayer Terms of Service

© 2025 SlidePlayer.com. Inc. All rights reserved.