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Fig. 6 Knockdown of nestin impaired tendon repair and regeneration in a rat model of patellar tendon defect. Knockdown of nestin impaired tendon repair.
Figure 4. Histological features of defect healing 7 weeks after defect creation (Masson trichrome staining, scale bar=1 mm). Group 1 (A) showed notable.
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Date of download: 10/24/2017 Copyright © ASME. All rights reserved. From: Effect of Implanting a Soft Tissue Autograft in a Central-Third Patellar Tendon Defect: Biomechanical and Histological Comparisons J Biomech Eng. 2011;133(9):091002-091002-6. doi:10.1115/1.4004948 Figure Legend: Force-displacement curves (mean ± SEM). PTA (n = 6) and NH (n = 6) repairs both exceeded the peak in vivo force required for activities of daily living (100 N; in vivo force and displacement, IVF and IVD, respectively) [,]. However, PTA and NH repairs do not match the normal central-third PT (Normal; n = 8 [,]) or TEC (n = 7 []) repair curves. Portions of this figure re-printed with permission from Juncosa-Melvin, (2006) [].

Date of download: 10/24/2017 Copyright © ASME. All rights reserved. From: Effect of Implanting a Soft Tissue Autograft in a Central-Third Patellar Tendon Defect: Biomechanical and Histological Comparisons J Biomech Eng. 2011;133(9):091002-091002-6. doi:10.1115/1.4004948 Figure Legend: Histological images of patellar and tibial tendon-to-bone insertion sites. Insertion site images are shown with H&E staining and immunohistochemical staining for type II collagen (Col2). After 12 weeks of healing, proper insertion sites were not regenerated by PTA or NH. The insertion sites in the native struts (NS) appear unaffected by the surgical procedure. H&E images are labeled to reflect: B, bone; T, tendon; FS, fibrous scar; MFC/FC, mineralized fibrocartilage/fibrocartilage zones of the insertion site, respectively (note: these zones are not as distinct in the patella as they are in the tibia); areas resembling neo-vascularization are marked with an asterisk. Scale bar = 200 μm.