Fig. 1. Infarct size and behavioral outcomes in response to single and repeated treatment of hUCB-MSCs after MCAO. (A) Schematic of the time schedule.

Slides:

Advertisements

Similar presentations

Figure 1.1 The observer in the truck sees the ball move in a vertical path when thrown upward. (b) The Earth observer views the path of the ball as a parabola.

Advertisements

Date of download: 6/25/2016 Copyright © 2016 SPIE. All rights reserved. Overview of the electrocorticogram-functional photoacoustic microscopy (ECoG-fPAM)

Fig. 2. The expression of pKr-2 in the substantia nigra (SN) of patients with Parkinson's disease (PD). (A, B) Western blot analysis for pKr-1-2 and pKr-2,

Fig. 4. miR-326, miR-330, and miR-3099 bound to the GFAP 3'-UTR and regulated GFAP expression. (A) Schematic diagram of potential binding sequence (seed.

Fig. 3. Expression of Ninj1protein in cortices of the ischemic hemispheres of the rat brain at 4 day post-MCAO. Coronal brain sections were prepared at.

Fig. 2. Neuroprotective effects of ex-4 through the G protein-coupled GLP-1 receptor. (A) Infarct volume was assessed by TTC staining after tMCAO in rats.

Fig. 4. TGF-α is one of the major cytokines in M2 conditioned media

Volume 9, Issue 2, Pages (February 2004)

Postischemic administration of liposome-encapsulated luteolin prevents against ischemia-reperfusion injury in a rat middle cerebral artery occlusion model

Fig. 1. A diagram of optical recording of intrinsic signal (ORIS) imaging in the MCAO model. (A) The middle cerebral artery occlusion (MCAO) was performed.

Fig. 1. In situ hybridization analysis for detecting endogenous mBest1 transcript in whole-brain region. In situ hybridization results from coronal (top.

Fig. 2. Post-treatment with isoflurane reduced infarct volume

Fig. 5. The measurement of phosphorylation of p53 level in N2A cells under high glucose condition and agmatine treatment. (A, B) p53 phosphorylation levels.

Cerebrovasc Dis 2016;42: DOI: /

Fig. 9. Treatment of flagellin increased cell survival in hypoxic conditions. (A) After 2 h OGD, cell cytotoxicity levels were measured by a LDH assay.

Fig. 2. Infection by lentivirus

Fig. 2. Expression of Ninj1 in cortices of ischemic hemispheres of rat brains at 1 day post-MCAO. Coronal brain sections were prepared at 1 day post-MCAO.

Fig. 2. Severities of seizures by durations and daily frequency at different time points. (A) An example of seizure activity in EEG recording. (B) Seizure.

Fig. 4. Reactive cerebral artery extraction using standard deviation (SD) map categorization is shown. (A) Image processing steps for the reactive artery.

12/15-Lipoxygenase Inhibition or Knockout Reduces Warfarin-Associated Hemorrhagic Transformation After Experimental Stroke by Yu Liu, Yi Zheng, Hulya Karatas,

Neuroprotective Effect of Darbepoetin Alfa, a Novel Recombinant Erythropoietic Protein, in Focal Cerebral Ischemia in Rats by Ludmila Belayev, Larissa.

L. Tong, M. Cai, Y. Huang, H. Zhang, B. Su, Z. Li, H. Dong

Oral contraceptive administration aggravates nitric oxide synthesis inhibition-induced high blood pressure in female rats L.A. Olatunji, A.O. Soladoye

Neuroprotection from Stroke in the Absence of MHCI or PirB

Performance of chronic METH-treated rats in the gambling test under the double-reward condition. Performance of chronic METH-treated rats in the gambling.

Asynchronous administration of xenon and hypothermia significantly reduces brain infarction in the neonatal rat J.L. Martin, D. Ma, M. Hossain, J. Xu,

Fig. 1. Mitochondrial internalization in cardiomyocytes.

Fig. 7. Lhx1-RNAi reduces the eye size

Fig. 6. Mucus analysis of lungs from allergic and tolerized mice

Fig. 4. Perinuclear dynein regulators are not required for primary ciliogenesis.RPE cells were transfected with siRNA and either nocodazole-treated, fixed,

Fig. 3. Mice which experienced MS or MSEW did not exhibit impairments in hippocampal and amygdala-dependent fear-related emotional memories. In the acquisition.

Fig. 4. Reduction of apoptosis and autophagic stress by treatment with silibinin in the KA-treated hippocampus. (A, B) Double-immunofluorescence staining.

Nutrient availability regulates growth of the X. laevis optic tectum.

Fig. 3. Neuroprotective effects of silibinin against KA-induced excitotoxicity. (A) Silibinin was intraperitoneally injected for 8 days, starting 1 day.

Fig. 5. Anti-inflammatory effects of silibinin on KA treatment in the hippocampus. (A) Representative coronal sections of the hippocampal CA1 region following.

Fig. 1. MS and MSEW mice did not exhibit depression-related behavior compared to controls. There was no significant difference in the time spent stationary.

Fig. 3. OPNpt20 binding to αv integrin in BV2 cells

Fig. 2. High density EEG of anterior and posterior spindles

Fig. 2. Induction of F-actin polymerization by OPNpt20

Fig. 3. Relationship between the generalization of conscious feeling induced by fear conditioning and individual anxiety (or depression) level. To determine.

Fig. 4. Activations of FAK, Erk, and Akt pathways by OPNpt20

Fig. 3. Comparing between source localization algorithms

Fig. 2. Infarct size and mNSS, weight change of animals with oral gavage of HSA and saline. (A) TTC staining of the individual brain was shown that the.

Fig. 3. Inhibition of HIF1α with chrysin induced cell death and suppressed the expressions of glycolysis-regulating genes in GBMs. (A~D) Cell viability.

Fig. 1. Changes in hippocampal matrix metalloproteinase-9 (MMP-9) and angiopoietin-2 (ANG-2) expression over time following bilateral common carotid artery.

Fig. 2. Scriptaid and RGFP966 alter macrophage phenotype

Fig. 5. Subcellular distribution (soma, process, and microdomain) of MOR in astrocyte (indicated in blue). MOR is stained with immunogold with silver enhancement.

Fig. 1. Schematic diagram of behavioral procedures

Fig. 1. HFD-induced obesity in C58BL/6 mice and decrease of general and motor behavior in HFD-fed mice. (A) HFD-fed, 5-week-old C57BL/6 mice (n=6/group)

Fig. 2. TH expression is decreased in mice with HFD-induced obesity

Fig. 1. Relationship between obesity and atopic dermatitis

Fig. 1. Induction of microglial cell motility by OPNpt20

Fig. 2. Disease state-specific alteration of activity-dependent BDNF secretion from cortical axons. (A) Average fluorescence changes with time (ΔFt/F0)

Fig. 4. Chronic microwave treatment produced changes in glucose and corticosterone levels in blood. (A) Blood glucose levels of mice subjected to chronic.

Fig. 10. Summary of astrocyte specificity and coverage of hGFAP-CreERT2 in various brain regions. (A) Schematic sagittal section diagram shows heterogeneous.

Fig. 7. Scriptaid reduces the formation of foamy macrophages of the M1 phenotype, whereas RGFP966 reduces the formation of foamy macrophages regardless.

Fig. 3. KMS99220 inhibits activation of NFκB signaling via HO-1 induction. (A) BV2 cells were treated with 10 µM KMS99220 for 1 h, and then treated with.

Fig. 4. A theoretical model of a neural interfacing system to recover neural functions after brain injuries by neuro-prosthetic treatments. A schematic.

Fig. 3. LPS induces degeneration of DA neurons and microglial activation in the SN in vivo. LPS (5 µg/3 µl) was unilaterally injected into the SN in the.

Fig. 8. Scriptaid and RGFP966 have no effect on functional recovery, lesion size or demyelinated area after SCI. BALB/c mice were subjected to a T-cut.

Fig. 9. Scriptaid and RGFP966 have no effect on Iba-+1 and CD4+ cell immune cell infiltration after SCI. BALB/c mice were subjected to a T-cut hemisection.

Fig. 3. Ultra-wide-field color FP and AF, spectral-domain OCT, and H&E staining images at one month after intravitreal MNU injection with pars plana vitrectomy.

Fig. 2. Ultra-wide-field color FP, AF, histology with H&E staining, and OCT images at one month after intravitreal injection of MNU without vitrectomy.

Fig. 3. hALDH1L1 promoter-driven Cre recombinase-mediated tdTomato expression in BLA. (A) Low magnification images of injection area in amygdala. Each.

Fig. 8. Electron micrographs taken from 8-week glaucoma retina

Fig. 7. Apoptotic cell death was evaluated via TUNEL

Fig. 1. The brain of Octopus minor. (A) Left: a live specimen of O

Fig. 5. Phagocytosis of zymosan particles by BV2 cells

Fig. 1. Genetic confimation of hGFAP-CreERT2 mouse line and its tamoxifen inducible Cre expression. (A) Genetic map of hGFAP-CreERT2. (B) The primer sequences.

Volume 9, Issue 2, Pages (February 2004)

Presentation transcript:

Fig. 1. Infarct size and behavioral outcomes in response to single and repeated treatment of hUCB-MSCs after MCAO. (A) Schematic of the time schedule. (B) Rotarod test. (C) Limb placement test (LPT). Data are means±SEM. control (n=11) vs D-2 (n=7) vs D-2+7 (n=5) group. (D) Infarct size in rats that received one or repeated hUCB-MSC treatment. Representative cresyl violet staining images (control group, left; one treat group, middle; repeat group, right). (E) Data are mean percentage of the infarct size±SEM, relative to the size of the intact hemisphere. *p<0.05, **p<0.01, control, D-2 and D-2+7 groups. Scale bar=1,000 µm. Fig. 1. Infarct size and behavioral outcomes in response to single and repeated treatment of hUCB-MSCs after MCAO. (A) Schematic of the time schedule. (B) Rotarod test. (C) Limb placement test (LPT). Data are means±SEM. control (n=11) vs D-2 (n=7) vs D-2+7 (n=5) group. (D) Infarct size in rats that received one or repeated hUCB-MSC treatment. Representative cresyl . . . Exp Neurobiol. 2017 Feb;26(1):55-65. https://doi.org/10.5607/en.2017.26.1.55