Stimulation-Induced Transient Nonmotor Psychiatric Symptoms following Subthalamic Deep Brain Stimulation in Patients with Parkinson's Disease: Association.

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Stimulation-Induced Transient Nonmotor Psychiatric Symptoms following Subthalamic Deep Brain Stimulation in Patients with Parkinson's Disease: Association with Clinical Outcomes and Neuroanatomical Correlates Stereotact Funct Neurosurg 2016;94:93-101 - DOI:10.1159/000445076 Fig. 1. Change (baseline to 24-week visit) in UPDRS (a), LEDD (b), BDI (c), HAM-D (d), YMRS (e) and Q-LES (f) scores in TNM+ and TNM- patients. Data presented as means ± SEM. * p < 0.05 by the two-tailed paired t test. © 2016 S. Karger AG, Basel

Stimulation-Induced Transient Nonmotor Psychiatric Symptoms following Subthalamic Deep Brain Stimulation in Patients with Parkinson's Disease: Association with Clinical Outcomes and Neuroanatomical Correlates Stereotact Funct Neurosurg 2016;94:93-101 - DOI:10.1159/000445076 Fig. 2. Neuroanatomical localization of active contacts. The spatial location of the active contacts on postoperative MR images fused with the Schaltenbrand and Wahren human brain atlas. a Illustration demonstrating the anatomical location of the active contacts in relation to surrounding structures: 1 = corticospinal tract; 2 = zona incerta; 3 = thalamus; 4 = putamen; 5 = globus pallidus external segment; 6 = globus pallidus internal segment; 7 = subthalamic nucleus; 8 = substantia nigra. b Serial sections of the STN corresponding to slices in the Schaltenbrand and Wahren human brain atlas on the left (L6-L17) and right (R6-R17) showing the locations of active contacts and corresponding TNM categories. M = Medial; L = lateral. © 2016 S. Karger AG, Basel