Volume 61, Issue 1, Pages (January 2002)

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Volume 61, Issue 1, Pages 61-67 (January 2002) Role of TGF-β1 in renal parenchymal scarring following childhood urinary tract infection Shirley A. Cotton, Rasheed A. Gbadegesin, Shelley Williams, Paul E.C. Brenchley, Nicholas J.A. Webb Kidney International Volume 61, Issue 1, Pages 61-67 (January 2002) DOI: 10.1046/j.1523-1755.2002.00110.x Copyright © 2002 International Society of Nephrology Terms and Conditions

Figure 1 Transforming growth factor-β1 (TGF-β1) production by peripheral blood mononuclear cells (PBMC); a comparison of TGF-β1 -800 genotype. TGF-β1 production was significantly higher in those patients with a GG -800 TGF-β1 genotype than those with a GA genotype. The thick horizontal bar denotes the median, and whiskers show the interquartile range. P = 0.031 by the Mann-Whitney U test. Kidney International 2002 61, 61-67DOI: (10.1046/j.1523-1755.2002.00110.x) Copyright © 2002 International Society of Nephrology Terms and Conditions

Figure 2 TGF-β1 production by PBMC; a comparison of the renal parenchymal scarring (RPS) versus the no scarring (NS) groups of children. There was no significant difference in PBMC TGF-β1 production between those patients who had not developed RPS (NS) and those who had developed RPS. The thick horizontal bar denotes the median, and whiskers show the interquartile range. P = 0.198 by the Mann-Whitney U test. Kidney International 2002 61, 61-67DOI: (10.1046/j.1523-1755.2002.00110.x) Copyright © 2002 International Society of Nephrology Terms and Conditions