Figure 3 Energy metabolism regulation, cardiovascular and bone disease in CKD Figure 3 | Energy metabolism regulation, cardiovascular and bone disease.

Slides:



Advertisements
Similar presentations
A.M. Thompson, T.G. Pickering  Kidney International 
Advertisements

Endocrine System.
Figure 1 Disruption of phosphate homeostasis in chronic kidney disease (CKD) Figure 1 | Disruption of phosphate homeostasis in chronic kidney disease (CKD).
Volume 67, Pages S1-S7 (June 2005)
CKD–Mineral and Bone Disorder: Core Curriculum 2011
Figure 5 A layered approach to the follow-up of patients with acute kidney disease (AKD) Figure 5 | A layered approach to the follow-up of patients with.
Figure 4 Interplay between acute kidney injury (AKI),
Figure 6 Effects of adiponectin on podocyte function
Figure 5 Inter-relationships between sleep apnoea, CKD and brain injury Figure 5 | Inter-relationships between sleep apnoea, CKD and brain injury. In chronic.
Figure 6 Differences in glycaemic control with the study drug
Figure 4 Interactions between adipose, the microbiome and kidney
Nat. Rev. Nephrol. doi: /nrneph
Figure 1 Mechanisms of kidney injury in the setting of obesity
Figure 1 Types of coronary artery calcification
Figure 2 Proinflammatory mechanisms in CKD
Figure 1 Circadian changes in energy metabolism and immune responses in CKD Figure 1 | Circadian changes in energy metabolism and immune responses in CKD.
Figure 1 Role of the kidney in glucose homeostasis
Figure 3 The fat–intestine–kidney axis
Nat. Rev. Nephrol. doi: /nrneph
Is it Important to Prevent and Treat Protein-Energy Wasting in Chronic Kidney Disease and Chronic Dialysis Patients?  Bereket Tessema Lodebo, MD, MPH,
Fibroblast Growth Factor 23 and CKD Prognosis
How do cells talk to each other
Figure 6 The bioavailability of phosphate differs according to the protein source Figure 6 | The bioavailability of phosphate differs according to the.
Figure 7 The efficacy of phosphate-binder therapy
Volume 79, Issue 9, Pages (May 2011)
Nat. Rev. Endocrinol. doi: /nrendo
Figure 5 Risk factor control in the intensive treatment group
Figure 2 The network of chronic diseases and their mutual influences
Figure 2 Three distinct mechanisms of activation of
Figure 1 The burden of chronic kidney disease (CKD)
CKD–Mineral and Bone Disorder: Core Curriculum 2011
FGF23 or PTH: which comes first in CKD ?
Figure 1 Acute kidney injury and chronic kidney disease
Figure 4 The gut–kidney axis, inflammation and cardiovascular disease in CKD Figure 4 | The gut–kidney axis, inflammation and cardiovascular disease in.
Figure 2 The continuum of acute kidney injury (AKI),
Figure 4 Model of changes in the serum levels
Ergocalciferol and Cholecalciferol in CKD
Figure 5 Consequences of CKD on lipid metabolism
Figure 5 Potential roles of phosphate and fibroblast growth factor 23 (FGF-23) in the development of cardiovascular disease in patients with chronic kidney.
Nat. Rev. Nephrol. doi: /nrneph
Figure 1 Specificity of the various epidermal growth factor (EGF)
Figure 2 Organ crosstalk in the pathophysiology
Nat. Rev. Nephrol. doi: /nrneph
John P. Middleton, Patrick H. Pun  Kidney International 
Nat. Rev. Nephrol. doi: /nrneph
Nat. Rev. Nephrol. doi: /nrneph
Nat. Rev. Nephrol. doi: /nrneph
M. Fukagawa, S. Nakanishi, J.J. Kazama  Kidney International 
Figure 1 Distinct features of adipocytes
Nat. Rev. Cardiol. doi: /nrcardio
Figure 3 Serum phosphate level is associated with
Nat. Rev. Nephrol. doi: /nrneph
Tally Naveh-Many, Justin Silver  Kidney International 
Volume 79, Pages S20-S23 (April 2011)
Figure 2 Mechanisms of crosstalk between adipocytes and the kidney
Nat. Rev. Nephrol. doi: /nrneph
Use of vitamin D in chronic kidney disease patients
Vitamin D receptor: A highly versatile nuclear receptor
Figure 3 Preventive strategies for CSA-AKI
Figure 1 Worldwide distribution of disease burden attributable to environmental risks in 2012 Figure 1 | Worldwide distribution of disease burden attributable.
Volume 87, Issue 3, Pages (March 2015)
Fibroblast growth factor 23: the making of a hormone
Nat. Rev. Nephrol. doi: /nrneph
Pablo A. Ureña Torres, Marc De Broe  Kidney International 
Mineral metabolism in CKD: adaptation devolves into maladaptation.
Nat. Rev. Nephrol. doi: /nrneph
The ins and outs of phosphate homeostasis
Diagram of the mechanisms involved in limiting the ability of the kidney to maintain the levels of 1,25-dihydroxyvitamin D in chronic kidney disease (CKD).
Marta Christov, Harald Jüppner  Kidney International 
Volume 86, Issue 2, Pages (August 2014)
Presentation transcript:

Figure 3 Energy metabolism regulation, cardiovascular and bone disease in CKD Figure 3 | Energy metabolism regulation, cardiovascular and bone disease in CKD. Altered levels of several molecules such as bone morphogenetic protein (BMP), 1,25-dihydroxyvitamin D, parathyroid hormone (PTH), fibroblast growth factor 23 (FGF-23), insulin, uraemic toxins and cardiac natriuretic peptides, as well as dysfunctional processes such as altered energy balance, adipocyte function, blood pressure and volume control are key risk factors for chronic kidney disease (CKD), bone disease and cardiovascular disease. Zoccali, C. et al. (2017) The systemic nature of CKD Nat. Rev. Nephrol. doi:10.1038/nrneph.2017.52