Cellular mechanisms of renal osteodystrophy

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Cellular mechanisms of renal osteodystrophy Judith A. Hoyland, Michael L. Picton  Kidney International  Volume 56, Pages S8-S13 (December 1999) DOI: 10.1046/j.1523-1755.1999.07312.x Copyright © 1999 International Society of Nephrology Terms and Conditions

Figure 1 PTH1R and TGF-β mRNA expression. Expression of PTH1R mRNA by osteoblasts in (a) renal hyperparathyroid bone and (b) in normal fracture callus. Note the increase in signal seen in nonuremic osteoblasts compared to those in renal bone. (c) PTH1R mRNA expression by osteoclasts in hyperparathyroid bone. (d) PTH1R mRNA expression by osteoclasts in pagetic bone. (e) TGF-β mRNA expression in renal hyperparathyroid bone in osteoblasts. (f) TGF-β mRNA expression in renal hyperparathyroid bone in osteoclasts. Kidney International 1999 56, S8-S13DOI: (10.1046/j.1523-1755.1999.07312.x) Copyright © 1999 International Society of Nephrology Terms and Conditions

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