Cardiovascular Emergencies

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Presentation transcript:

Cardiovascular Emergencies Chapter 14 Cardiovascular Emergencies

Cardiovascular Emergencies Cardiovascular disease has been leading killer of Americans since 1900. Accounts for 1 of every 2.8 deaths Cardiovascular disease (CVD) claimed 931,108 lives in the US during 2001. 2,551 per day Almost two people per minute!

Prevention EMS can help reduce deaths by: Encouraging healthy life-style Early access More CPR training of laypeople Public access to defibrillation devices Recognizing need for advanced life support (ALS)

Physiology of the Circulatory System (1 of 2) Pulse: The wave of blood through the arteries formed when the left ventricle contracts Can be felt where an artery passes near the skin surface and over a bone

Physiology of the Circulatory System (2 of 2) Blood pressure: Amount of force exerted against walls of arteries Systole: Left ventricle contracts Diastole: Left ventricle relaxes Perfusion: Circulation of blood within an organ or tissue If inadequate, the patient goes into shock

Pulse Points Corotid Femoral Brachial Radial Posterior Tibial Dorsalis Pedis

Components 1. Heart (pump) 2. Vessels (pipes) 3. Blood (fluid)

The Heart Muscular organ Myocardium--the heart muscle Lies directly behind the sternum Has 4 chambers -Left and right atriums (upper chambers) -Left and right ventricles (lower chambers) Septum--divides left and right sides

The Heart Has it’s own blood supply Has it’s own electrical system The heart is divided into 2 types of circulation: 1. Pulmonary Circulation 2. Systemic Circulation

Pulmonary Circulation Circulation to and from lungs Right side of heart Unoxygenated blood enters the right atrium from the superior vena cava Enters the right ventricle through a valve Pumped through pulmonary arteries to lungs Receives oxygen from alveoli and leaves waste products and carbon dioxide

Systemic Circulation Circulation to and from body Left side of heart Oxygenated blood enters left atria through pulmonary vein Enters left ventricle through a valve Pumped to aorta and the body Delivers oxygen to cells and removes waste

Cardiovascular Structure and Function

Blood Flow Through the Heart 1. Vena Cava 2. Right Atrium 3. Tricuspid valve 4. Right Ventricle 5. Pulmonic Valve 6. Pulmonary Artery 7. Lungs 8. Pulmonary Vein 9. Left Atrium 10. Mitral Valve 11. Left Ventricle 12. Aortic Valve 13. Aorta 14. Body

Blood Flow Through the Heart

Electrical System of the Heart

Coronary Arteries

Blood Vessels Arteries--Carries blood away from the heart (usually oxygenated) Arteries branch into arterioles and then into capillaries Veins--Carries blood toward the heart (usually unoxygenated) Veins branch into venules and then into capillaries

Blood Vessels Capillaries--Very thin vessels where the actual gas exchanges occurs -oxygen and nutrients delivered to cells -carbon dioxide and waste picked up Forms capillary beds

Blood Components Plasma -sticky yellowish -carries blood cells and nutrients Erythrocytes (red blood cells) -contain iron -gives blood it’s color -carries oxygen

Blood Components Leukocytes (white blood cells) -fights infection Platelets -clot formation -usually clots in 4 to 6 minutes

Cardiac Compromise Chest pain results from ischemia Ischemic heart disease involves decreased blood flow to the heart. If blood flow is not restored, the tissue dies.

Coronary Artery Disease (CAD) Atherosclerosis Arteriosclerosis

Atherosclerosis Materials build up inside blood vessels. This decreases or obstructs blood flow. Risk factors place a person at risk.

Arteriosclerosis Coronary artery wall becomes hard and stiff due to calcium deposits “Hardening of the Arteries” Causes coronary arteries to loose their elastic nature Decreases blood flow Causes hypertension

Major Risk Factors of Coronary Artery Disease Uncontrollable Age Sex Race Heredity Controllable Smoking High Blood Pressure High Cholesterol Diabetes

Minor Risk Factors of Coronary Artery Disease Obesity Inactivity Stress Personality

Coronary Artery Disease

Coronary Artery Disease

Angina Pectoris Pain in chest that occurs when the heart does not receive enough oxygen Typically crushing or squeezing pain Rarely lasts longer than 10-15 minutes Can be difficult to differentiate from heart attack

Angina Pectoris Signs/Symptoms 1. Squeezing/crushing chest pain 2. Pain may radiate to shoulders, arms, neck, jaw, upper back, or upper abdomen 3. Pain may be in area of radiation only 4. Possible shortness of breath (SOB)

Angina Pectoris 5. Pain associated with the “3 E’s” -Exercise -Eating -Emotion 6. Pain seldom lasts for more than 10-15 minutes 7. Pain normally relieved by: -rest -nitroglycerin

Angina Pectoris Following an angina attack, there is NO residual damage to the myocardium

Angina Pectoris Two Forms of Angina 1. Stable Angina -Pain predictable in duration and frequency -Pain relieved by predictable amounts of rest and nitroglycerin 2. Unstable Angina -Change occurs in usual patterns -30% go on to infarct within 3 months

Treatment of Angina Pectoris Treat all first time angina and unstable angina as a myocardial infarction. When in doubt, manage all chest pain as a myocardial infarction

Acute Myocardial Infarction Acute Myocardial Infarction (AMI)--Heart Attack Death of the myocardium due to inability of diseased coronary arteries to allow adequate perfusion Once myocardium tissue dies, it will not regenerate

Myocardial Infarction Incidence MI is the leading cause of death in the US 1 to 1.5 million Americans will have a MI this year. Of these, about 600,000 will die. 350,000 will die in the first 2 hours after symptoms begin, without ever reaching the hospital!!

Acute Myocardial Infarction (AMI) Pain signals death of cells. Opening the coronary artery within the first hour can prevent damage. Immediate transport is essential.

Pain of AMI Chest Pain is “cardinal sign” of an AMI Occurs in 85% of AMIs May or may not be caused by exertion Does not resolve in a few minutes Can last from 30 minutes to several hours May not be relieved by rest or nitroglycerin

Pain of AMI May be crushing, squeezing, tight, heavy May radiate to neck, jaw, shoulders, arm, upper back, or even abdomen May occur in areas of radiation only May vary in intensity, unaffected by swallowing, coughing, deep breathing, or movement 15% have “silent AMI”

Signs/Symptoms of AMI Chest pain Shortness of breath (SOB) Weakness, dizziness, fainting Nausea, vomiting Pallor, diaphoresis (sweating) Feeling of impending doom Pulmonary Edema

Signs/Symptoms of AMI Changes is pulse, BP, or respirations are not diagnostic of an AMI Early recognition is critical 50% of deaths occur in first 2 hours. But the average person waits 3 hours before seeking help

Sudden Death 40% of AMI patients do not reach the hospital. Death is due to Arrhythmias (irregular heart rhythm) Heart may be twitching.

Arrhythmias (1 of 2) Bradycardia Ventricular Tachycardia

Arrhythmias (2 of 2) Ventricular Tachycardia Ventricular Fibrillation Asystole

Treatment of Cardiac Chest Pain (1 of 3) 1. Reassure Pt, High concentration of oxygen 2. Give aspirin 3. Reassure/calm patient 4. Obtain brief history and perform physical exam 5. Give nitroglycerin

Treatment of Cardiac Chest Pain (2 of 3) 6. If patient has history of angina with changes in pattern, transport immediately. 7. Transport in semi-sitting position if BP is normal, supine if BP is low. 8. Do NOT allow patient to walk to ambulance. 9. Don’t use lights and sirens if patient is awake, alert, and breathing without distress.

Treatment of Cardiac Chest Pain (3 of 3) 10. Monitor vital signs every 10 minutes 11. Request ALS Backup -90% of deaths occur from arrhythmias. -Arrhythmias can be treated with early drug therapy. 12. ALWAYS examine for pedal edema and listen to lung sounds

Cardiogenic Shock Heart lacks power to force blood through the circulatory system. Onset may be immediate or not apparent for 24 hours after AMI. Failure of the heart and circulatory system Low BP Arrhythmias: Irregular heart beats

Congestive Heart Failure Congestive Heart Failure (CHF)--Inability of the heart to pump blood out as fast as it enters. Can be left-sided or right-sided Usually begins with left-sided failure.

Congestive Heart Failure Causes of CHF Coronary Artery Disease (CAD) Chronic hypertension MI Valvular heart disease

Congestive Heart Failure Pathophysiology Left ventricle fails Blood backs up into lungs Pressure increases in capillary beds Fluids forced out of capillary beds into the alveoli causing pulmonary edema; fluid in the lungs

Congestive Heart Failure Signs/Symptoms Dyspnea on exertion Paroxysmal nocturnal dyspnea Orthopnea-dyspnea lying down Tachycardia-rapid pulse rate (>100 bpm)

Congestive Heart Failure Signs/Symptoms (Cont.) Tachypnea-rapid breathing Noisy, labored breathing Rales, wheezing Pink, frothy sputum

Congestive Heart Failure Right sided failure most commonly caused by left sided failure. Blood backs up into systemic circulation -distended neck veins -fluid in abdominal cavity -pedal edema-fluid in feet and ankles

Congestive Heart Failure Treatment Sit patient up with feet down Hi concentration of oxygen Assist ventilations as needed Monitor vital signs every 5 to 10 minutes Request ALS backup

Hypertensive Emergencies (1 of 2) Systolic pressure greater than 160 mm Hg Common symptoms include altered mental status and pulmonary edema. If untreated, can lead to stroke or dissecting aortic aneurysm. Common symptoms Sudden, severe headache Strong, bounding pulse Ringing in the ears

Hypertensive Emergencies (2 of 2) Common symptoms Nausea and vomiting Dizziness Warm skin (dry or moist) Nosebleed Normally, there is no pre-hospital care for hypertension at the EMT-B level Rapid transport, contact Paramedic backup

Aortic Aneurysm Aortic aneurysm is weakness in the wall of the aorta. Susceptible to rupture Dissecting aneurysm occurs when inner layers of aorta become separated Primary cause: uncontrolled hypertension

S/S of Aortic Aneurysm Very sudden chest pain Comes on full force Different blood pressures between extremities May complain of flank pain Transport patients quickly and safely.

Physical Findings of Cardiac Compromise Pulse rate increases and may be irregular. Blood pressure may be normal or falling. Respirations are usually normal. General appearance Frightened Nausea, vomiting, cold sweat

Approach to the Patient with Chest Pain (1 of 2) Reassure the patient and perform initial assessment. Administer oxygen. Measure and record vital signs. Place the patient in a position of comfort.

Approach to the Patient with Chest Pain (2 of 2) Obtain history and physical exam. Ask about the chest pain Assist with administration of prescribed nitroglycerin. Transport promptly. Report to medical control en route.

Heart Surgeries Coronary artery bypass graft (CABG) Angioplasty Cardiac pacemaker

Internal Cardiac Pacemakers Maintains a regular heart rhythm and rate Do not place AED patches over pacemaker.

Automatic Implantable Cardiac Defibrillators Monitor heart rhythm and deliver shocks as needed. Low electricity will not affect rescuers.