Pancreas and Liver Injury Are Associated in Individuals With Increased Alcohol Consumption  Andrea Pace, Andreas de Weerth, Marc Berna, Katharina Hillbricht, Michael Tsokos, Michael Bläker, Klaus Pueschel, Ansgar W. Lohse  Clinical Gastroenterology and Hepatology  Volume 7, Issue 11, Pages 1241-1246 (November 2009) DOI: 10.1016/j.cgh.2009.06.010 Copyright © 2009 AGA Institute Terms and Conditions

Figure 1 Age distribution at time of death of the study population (n = 620). Clinical Gastroenterology and Hepatology 2009 7, 1241-1246DOI: (10.1016/j.cgh.2009.06.010) Copyright © 2009 AGA Institute Terms and Conditions

Figure 2 Causes of death of the study population (n = 620). Clinical Gastroenterology and Hepatology 2009 7, 1241-1246DOI: (10.1016/j.cgh.2009.06.010) Copyright © 2009 AGA Institute Terms and Conditions

Figure 3 Incidence of various degrees of liver disease in the study population (n = 620). Clinical Gastroenterology and Hepatology 2009 7, 1241-1246DOI: (10.1016/j.cgh.2009.06.010) Copyright © 2009 AGA Institute Terms and Conditions

Figure 4 Incidence of various degrees of pancreatic disease in the study population (n = 620). Clinical Gastroenterology and Hepatology 2009 7, 1241-1246DOI: (10.1016/j.cgh.2009.06.010) Copyright © 2009 AGA Institute Terms and Conditions

Figure 5 Incidence of various degrees of liver disease (A–D) in patient groups with a given pancreatic change. *P < .05, **P < .01, ***P < .001, compared with the pancreas 0 group. Clinical Gastroenterology and Hepatology 2009 7, 1241-1246DOI: (10.1016/j.cgh.2009.06.010) Copyright © 2009 AGA Institute Terms and Conditions

Figure 6 Incidence of various degrees of pancreatic disease (A–D) in patients groups with a given liver change. **P < .01, ***P < .001, compared with the liver 0 group. Clinical Gastroenterology and Hepatology 2009 7, 1241-1246DOI: (10.1016/j.cgh.2009.06.010) Copyright © 2009 AGA Institute Terms and Conditions