Cerebral Vasospasm After Subarachnoid Hemorrhage DOUGLAS CHYATTE, M.D.  Mayo Clinic Proceedings  Volume 59, Issue 7, Pages 498-505 (July 1984) DOI: 10.1016/S0025-6196(12)60441-8 Copyright © 1984 Mayo Foundation for Medical Education and Research Terms and Conditions

Fig. 1 Electron micrograph of a canine basilar artery, showing normal appearance of endothelial cells (E) adjacent to vessel lumen (L), elastic lamina (EL), and smooth muscle cells (S). (× 3,000.) Mayo Clinic Proceedings 1984 59, 498-505DOI: (10.1016/S0025-6196(12)60441-8) Copyright © 1984 Mayo Foundation for Medical Education and Research Terms and Conditions

Fig. 2 Electron micrograph of a chronically vasospastic canine basilar artery, showing striking necrosis of smooth muscle cells (S). Ultrastructural abnormalities involving endothelium (E) and elastic lamina (EL) are also apparent, (× 1,950.) Mayo Clinic Proceedings 1984 59, 498-505DOI: (10.1016/S0025-6196(12)60441-8) Copyright © 1984 Mayo Foundation for Medical Education and Research Terms and Conditions

Fig. 3 Bar graph, comparing contractile response of isolated normal (control) and vasospastic canine basilar artery rings. 5HT = 5-hydroxy-tryptamine; PGF2a = prostaglandin F2. Mayo Clinic Proceedings 1984 59, 498-505DOI: (10.1016/S0025-6196(12)60441-8) Copyright © 1984 Mayo Foundation for Medical Education and Research Terms and Conditions