Chronic ocular ischemia and neovascular glaucoma: A result of extracranial carotid artery disease Willis H. Wagner, MD, Fred A. Weaver, MD, James R. Brinkley, MD, Mark Borchert, MD, Steven F. Lindsay, MD Journal of Vascular Surgery Volume 8, Issue 5, Pages 551-557 (November 1988) DOI: 10.1016/0741-5214(88)90304-7 Copyright © 1988 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions
Fig. 1 A, Fluorescein angiogram of the left eye shows delayed filling of the retinal veins and patchy nonperfusion of the underlying choroid. B, Normal fluorescein angiogram, for comparison, demonstrates filling of both retinal arteries and veins at a time after injection similar to that in A. The area of choroidal hypofluorescence is due to normal blockage of fluorescence by macular pigment. Journal of Vascular Surgery 1988 8, 551-557DOI: (10.1016/0741-5214(88)90304-7) Copyright © 1988 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions
Fig. 2 Selective arteriograms show high-grade (greater than 95%) stenoses of right (A) and left (B) internal carotid arteries. Journal of Vascular Surgery 1988 8, 551-557DOI: (10.1016/0741-5214(88)90304-7) Copyright © 1988 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions
Fig. 3 Example of iris neovascularization (not from described case) shows lacy network of superficial vessels on iris. (Courtesy of David E. Eifrig, MD, University of North Carolina.) Journal of Vascular Surgery 1988 8, 551-557DOI: (10.1016/0741-5214(88)90304-7) Copyright © 1988 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions