Figure 10. Recovery of the acetyl-CoA and ATP levels by glucose administration. Figure 10. Recovery of the acetyl-CoA and ATP levels by glucose administration.

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Volume 70, Issue 8, Pages (October 2006)

PDE5A is expressed by podocytes.

TGF-β1 induces ILK activity in renal tubular epithelial cells.

Relationship between urinary TNF-α and RANTES excretion

Differential expression of select miRNAs was validated in a second cohort of patients. Differential expression of select miRNAs was validated in a second.

Podocyte-specific PPAR-γ–deficient mice show increased glomerular TRPC6 expression. Podocyte-specific PPAR-γ–deficient mice show increased glomerular TRPC6.

Antibody–mediated C5 inhibition in hepatocyte-Cfh−/− mice during accelerated serum NTN. (A) Hematuria, (B) serum urea, (C) red cell fragments, (D) glomerular.

Two independent amino acid positions were identified after stepwise association analysis for amino acids in HLA-DRβ1, HLA-DQα1 and HLA-DQβ1 in the combined.

Figure 1. Relationship between (A) total antioxidative activity (TAA) and endothelium-dependent vasodilation (EDV) (r = 0.41, P = 0.016), (B) lag phase.

Missense mutations in TNXB as a cause of VUR

Figure 6. Representative micrographs showing macrophages and nitrotyrosine in atherosclerotic plaques in aortic root cross-sections from apo-E −/− mice.

Figure 3. Severity of aortic atherosclerosis in apo-E −/− mice with CRF. Aortic plaque area fraction (A), aortic cholesterol content (B), and aortic root.

Figure 2. Plasma lipoprotein cholesterol in apo-E −/− mice with CRF

Both patient 1 and her family members showed a normal physiological AQP2 response to DDAVP. Immunoblotting for AQP2 was performed on urinary exosomes derived.

Figure 1. Plasma urea concentrations in apolipoprotein E–deficient (apo-E −/−) mice with chronic renal failure (CRF). Figure 1. Plasma urea concentrations.

Studies reporting the incident rate for all types of infections per 1000 patient days. Studies reporting the incident rate for all types of infections.

Several microRNAs are differentially expressed in the glomeruli between different types of kidney disease. Several microRNAs are differentially expressed.

INT-767 treatment prevents the increase in renal inflammation and oxidative stress in diabetic DBA/2J mice. INT-767 treatment prevents the increase in.

INT-767 treatment prevents the increase in renal HIF and Glut1 expression and ER stress in diabetic DBA/2J mice. INT-767 treatment prevents the increase.

INT-767 treatment prevents renal lipid accumulation in diabetic DBA/2J mice. INT-767 treatment prevents renal lipid accumulation in diabetic DBA/2J mice.

Renal microvascular injury 6 months after induction of JG cell-specific Gsα knockout. Renal microvascular injury 6 months after induction of JG cell-specific.

The proportion of DCD donor kidneys discarded was correlated with the proportion of NBD donor kidneys discarded within DSAs (Pearson correlation coefficient.

The incidence of all cause graft failure, and death with a functioning graft was higher in patients who received a DCD donor transplant with total donor.

VEGF signals via a paracrine loop to VEGFR-2 in the glomerulus.

Model of human CLC-Kb transporter topology and positions of amino acid exchanges due to mutations in the CLCNKB gene as identified in Bartter syndrome.

Pioglitazone protects against aging-related renal injury.

SS-31 reduces inflammation after acute renal ischemia.

Classification by HtTKV0 and age at HtTKV0 predicts the change in eGFR over time in class 1 patients. Classification by HtTKV0 and age at HtTKV0 predicts.

The increased cytosolic and nuclear β-catenin in the tubular epithelial cells of 7-d obstructed kidneys is suppressed by recombinant sFRP4. The increased.

(A through C) Mean eGFR during follow-up according to treatment assignment in patients with normoalbuminuria (A), microalbuminuria (B), and macroalbuminuria.

Figure 4. p38α mitogen-activated protein kinase (MAPK) blockade reduces interstitial volume in obstructed kidneys but does not reduce the number of infiltrating.

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Aerobic respiration. Aerobic respiration. There are 4 major reaction pathways including glycolysis, the conversion of pyruvate to acetyl-CoA, the TCA cycle,

ILK knockdown increases the survival of PKD mice.

Figure 5. Tissue uptake of circulating 125I-β2-m.

Ciliary abnormalities of jck renal epithelia.

UMOD and ACSM2A expressions correlate with renal function.

The insulin tolerance test at week 18.

Figure 1. Genomic organization of the murine Pcln1 gene.

Vitamin K2 supplementation reduces dp-ucMGP but not dp-cMGP levels in dialysis patients. Vitamin K2 supplementation reduces dp-ucMGP but not dp-cMGP levels.

Box plots showing the relative difference in distribution of mtDNA copy number per cell between patients with ESRD and healthy control subjects frequency.

Representative photomicrographs of immunohistochemical staining for Wilms’ tumor 1 (WT1) and cleaved caspase-3 in mouse kidney sections at 10, 20, and.

Figure 3. Effect of spironolactone or albumin conjugation on the vasoconstrictor action of aldosterone in afferent arterioles (left) and efferent arterioles.

(A) Mean (SD) serum continuous erythropoietin receptor activator (C. E

(A) Glucose values (mean +SEM) during continuous glucose monitoring while consuming whey protein (solid lines and filled circles) or placebo (broken lines.

Adjusted means (±SD) of eGFR in relation to the histologic severity of nonalcoholic steatohepatitis (i.e., NASH/fibrosis stage increasing from 0 to 3)

Analysis of renal transcriptome responses identifies LX-regulated transcriptional networks. Analysis of renal transcriptome responses identifies LX-regulated.

Nrf2−/− mice suffered greater renal damage by STZ compared with Nrf2+/+ mice. Nrf2−/− mice suffered greater renal damage by STZ compared with Nrf2+/+ mice.

Renal units with renal scarring by vesicoureteral reflux (VUR) grade.

Ca2+ infusion rates during all three protocol versions.

Blood glucose levels in chicken embryos.

Effects of rhein on FBG (A) and glucose intolerance (B) in db/db mice.

A loading dose decreases the time to achieve the target concentration.

Effect of randomized treatment on all renal events (top) and the composite of all renal events, macrovascular events, or all-cause mortality (bottom) according.

Graph showing percentage of total patients with PTD or IGT that would be detected in each category of FBG if an oral glucose tolerance test were performed.

Unbound drug concentrations in plasma (dotted) and brain ISF (solid, calculated from recovery and dialysate concentration) following repeated subcutaneous.

Mortality caused by cardiovascular disease (A) and sepsis (B) of patients with end-stage renal disease (ESRD) treated by dialysis compared with the general.

New dialysis starts in the United States by year in patients with and without diagnosis of diabetes. New dialysis starts in the United States by year in.

Annual hospital charges and costs for CEA and CAS

Causal diagrams that represent three possible relationships between smoking, ESRD, and albumin-to-creatinine ratio (ACR) in the Study of Heart and Renal.

Separation in achieved BP levels between the two intervention groups in the SPRINT participants with CKD. The broken line and open circles denote the intensive.

Distribution of SDS for serum creatinine, serum BUN, SBP, and DBP per quintiles of KS. Quintiles are expressed in SDS (see Figure 1B): 1st quintile,

Bland-Altman plot of arterial and central venous blood Pco2 showing the regression line (solid line) and the 95% limits of agreement of −12.3 to 4.8 mmHg.

Receiver operator characteristic (ROC) curve for fasting blood glucose (FBG) predicting posttransplantation diabetes (PTD) using time 0 FBG (a) and screening.

Plateau 13C-ketoisocaproic acid (KIC) enrichment during whole-body protein turnover (WBPT) study. 13C-KIC values (mean ± SD) in 12 patients in moles percent.

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Figure 10. Recovery of the acetyl-CoA and ATP levels by glucose administration. Figure 10. Recovery of the acetyl-CoA and ATP levels by glucose administration. (A) Time course of acetyl-CoA level in renal cortex of the (−/−) mice after refeeding. (B) Time course of the ATP level in renal cortex of the (−/−) mice after refeeding. Solid and open circles show the level after the administration of 50% glucose and 22% tripalmitin, respectively. Values are presented as means ± SD (n = 5). **P < 0.01 in comparison with St. St., (− /−) second starvation. Yuji Kamijo et al. JASN 2002;13:1691-1702 ©2002 by American Society of Nephrology