Electrical Storm in the Brain and in the Heart: Epilepsy and Brugada Syndrome  Gabor Sandorfi, MD, Bela Clemens, MD, PhD, Zoltan Csanadi, MD, PhD  Mayo Clinic Proceedings  Volume 88, Issue 10, Pages 1167-1173 (October 2013) DOI: 10.1016/j.mayocp.2013.06.019 Copyright © 2013 Mayo Foundation for Medical Education and Research Terms and Conditions

Figure 1 A, Initial 12-lead electrocardiogram (ECG) transmitted from the patient’s home. B, The 12-lead ECG recorded on arrival at the hospital. Mayo Clinic Proceedings 2013 88, 1167-1173DOI: (10.1016/j.mayocp.2013.06.019) Copyright © 2013 Mayo Foundation for Medical Education and Research Terms and Conditions

Figure 2 A, Electrocardiograms showing ventricular tachycardia initiated during procainamide infusion and the type I Brugada electrocardiographic pattern after termination. B, Electrocardiograms showing initiation and spontaneous termination of ventricular tachycardia. Tachycardia was initiated with double extrastimuli. BPM = beats/min. Mayo Clinic Proceedings 2013 88, 1167-1173DOI: (10.1016/j.mayocp.2013.06.019) Copyright © 2013 Mayo Foundation for Medical Education and Research Terms and Conditions

Figure 3 Ictal electroencephalographic recording, longitudinal bipolar montage. The ictal activity begins as a rhythmic discharge in 2 left frontal derivations (time scale, 30:44). It quickly spreads to the parietal cortex bilaterally (time scale, 30:52). Later, slow activity is maximal over most of the left hemisphere, and less abnormal theta activity dominates over the remaining cortex (time scale, 31:01). Note that the amplification changes from the upper to the lower panel. Mayo Clinic Proceedings 2013 88, 1167-1173DOI: (10.1016/j.mayocp.2013.06.019) Copyright © 2013 Mayo Foundation for Medical Education and Research Terms and Conditions