Figure 5 Genotype-positive hypertrophic

Slides:

Advertisements

Similar presentations

Date of download: 6/3/2016 Copyright © The American College of Cardiology. All rights reserved. From: Pregnancy in Patients With Pre-Existing Cardiomyopathies.

Advertisements

Date of download: 6/24/2016 Copyright © The American College of Cardiology. All rights reserved. From: Diagnostic Value of CMR in Patients With Biomarker-Positive.

Date of download: 7/7/2016 Copyright © The American College of Cardiology. All rights reserved. From: Toward clinical risk assessment inhypertrophic cardiomyopathy.

Date of download: 7/7/2016 Copyright © The American College of Cardiology. All rights reserved. From: Frequency and clinical expression of cardiac troponin.

Nat. Rev. Cardiol. doi: /nrcardio

Two-dimensional echocardiogram from a patient with severe hypertrophic cardiomyopathy. There is a severe increase in left ventricular wall thickness, with.

A Case of Elderly Women with Biventricular Hypertrophic Cardiomyopathy

Volume 81, Issue 1, Pages (January 1982)

Figure 1 Initial management of a patient with acute heart failure

High Incidence of Cardiac Malformations in Connexin40-Deficient Mice

Figure 10 Assessment of myocardial fibrosis

New Developments in Hypertrophic Cardiomyopathy

Nat. Rev. Cardiol. doi: /nrcardio

Volume 9, Issue 1, Pages (January 2012)

Figure 1 Cine cardiovascular magnetic resonance (CMR)

Editorial Heart Failure Clinics

Figure 3 T1 mapping and late gadolinium enhancement (LGE)

Nat. Rev. Cardiol. doi: /nrcardio

Figure 3 Imaging characteristics of cardiac myxoma

Figure 6 Risk stratification in hypertrophic cardiomyopathy (HCM)

The First Cardiac Operation Using Cardiopulmonary Bypass

Nat. Rev. Cardiol. doi: /nrcardio

Figure 4 Imaging in patients with suspected acute coronary syndrome

Figure 3 Traditional geometry and spatial fractals

Figure 8 4D magnetic resonance imaging patterns

Figure 2 Echocardiographic methods to estimate left atrial pressure

Figure 6 Speckle tracking echocardiography to

Figure 4 Doppler echocardiographic assessment

Nat. Rev. Cardiol. doi: /nrcardio

Figure 2 Results of diastolic stress testing

Figure 10 Lipomatous hypertrophy of the atrial septum

Wretched Excess: Stool-softener Abuse and Cardiogenic Shock

Nat. Rev. Cardiol. doi: /nrcardio

Nat. Rev. Cardiol. doi: /nrcardio

Nat. Rev. Cardiol. doi: /nrcardio

Figure 10 The energy depletion hypothesis

Nat. Rev. Cardiol. doi: /nrcardio

Closure of muscular ventricular septal defects guided by en face reconstruction and pictorial representation Kothandam Sivakumar, DM, Sivadasan Radha.

Nat. Rev. Cardiol. doi: /nrcardio

Nat. Rev. Cardiol. doi: /nrcardio

Figure 2 Apical hypertrophy missed by echocardiography

A 45-year-old with pulmonary hypertension

Figure 6 Cardiac fibroma

Scan, plan, print, practice, perform: Development and use of a patient-specific 3- dimensional printed model in adult cardiac surgery Joshua L. Hermsen,

Figure 2 Cardiac metastases

The effect of GOODCOLL versus POORCOLL on LVEF, LVEDV and scar at 4 months. The effect of GOODCOLL versus POORCOLL on LVEF, LVEDV and scar at 4 months.

International Journal of Cardiology

Nat. Rev. Cardiol. doi: /nrcardio

Jacques A. M. van Son, MD. (by invitation), Gordon K

Figure 6 The haemodynamic effects of

Substrate mapping for unstable ventricular tachycardia

Nat. Rev. Cardiol. doi: /nrcardio

Hiroo Takayama, MD, PhD, Wendy K. Chung, MD, PhD, Mathew S

Nat. Rev. Cardiol. doi: /nrcardio

A new concept for correction of systolic anterior motion and mitral valve regurgitation in patients with hypertrophic obstructive cardiomyopathy Joerg.

Bar chart of systolic function in healthy individuals, HCM LVH− patients and HCM LVH+ patients. GLS was significantly worse in the HCM LVH+ patients compared.

Bar chart of cardiac volumes and EF in the healthy individuals, HCM LVH− patients and HCM LVH+ patients. Blue bars show the indexed diastolic volumes (EDVI.

Barlow disease: Simple and complex

Three-dimensional (3D) measurement of the outer and inner/effective mitral annular area. Three-dimensional (3D) measurement of the outer and inner/effective.

Tricuspid Valve Prolapse Diagnosed by Cross-Sectional Echocardiography

Fig. 1. Bj mutant exhibits outflow tract malalignment defects.

Examples of association between CMR features and abnormal Q waves.

Nat. Rev. Cardiol. doi: /nrcardio

Extended septal myectomy for hypertrophic obstructive cardiomyopathy with anomalous mitral papillary muscles or chordae Kenji Minakata, MD, Joseph A.

Recurrent polymorphic ventricular tachycardia initiated by Purkinje ectopy in a patient with cardiac sarcoidosis Faisal Matto, MD, Mohsan Chaudhry, MD,

Scatter plot of relationship between MWT, GLS and EF upper panel shows that GLS was closely related to MWT with worse LV function by GLS correlating to.

Illustrative four-chamber (left) and parasternal long axis (right) echocardiographic image of a female aged 49 years with systolic displacement of anterior.

Hugh D. White et al. JIMG 2013;6:

Illustration of discordance between location of Q waves on ECG derivations and location of LGE in the myocardium on CMR. (A) Incidence of LGE within cited.

Presentation transcript:

Figure 5 Genotype-positive hypertrophic cardiomyopathy (HCM) without left ventricular hypertrophy (LVH) Figure 5 | Genotype-positive hypertrophic cardiomyopathy (HCM) without left ventricular hypertrophy (LVH)116,117,118,119,120,121,122,123,124. Cardiac magnetic resonance (CMR) images shown are from individuals who carry mutations implicated in HCM but do not show LVH (upper row) and healthy controls (lower row). Structural abnormalities in so-called genotype-positive LVH-negative individuals include: a | clefts, often multiple (arrowheads); b | elongation of the anterior mitral valve leaflet (arrows); c | increased apical trabeculae; and d | abnormal septal curvature. In the normal heart, the position of the interventricular septum is either neutral or convex into the right ventricle (d, lower), whereas in HCM it is convex into the left ventricle (d, upper). Septal curvature is measured as the maximal distance from the left ventricular endocardial border (B) to an intersection point (A) with a theoretical perpendicular joining the mid-septal wall at tricuspid valve insertion level with the apical right ventricular insertion point. The A–B distance seems to be increased in mutation carriers (5.0 ± 2.5 mm versus 1.6 ± 2.4 mm; P ≤0.0001) versus matched controls124. Sen-Chowdhry, S. et al. (2016) Update on hypertrophic cardiomyopathy and a guide to the guidelines Nat. Rev. Cardiol. doi:10.1038/nrcardio.2016.140