Figure 1. Prebiopsy and postbiopsy MRI

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Neurology Resident and Fellow Section

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Silent Brain Infarcts by Yi-Cheng Zhu, Carole Dufouil, Christophe Tzourio, and Hugues Chabriat Stroke Volume 42(4): March 28, 2011 Copyright ©

Figure 2. A patient with multifocal nodular lesions diagnosed with CNS tuberculosis A patient with multifocal nodular lesions diagnosed with CNS tuberculosis.

Figure 4 Paradoxical immune reconstitution inflammatory syndrome

Figure 1 Initial brain imaging (A–C) patient 1; (D–F) patient 2; (G–I) patient 3; (J–L) patient 4; and (M) patient 2. Initial brain imaging (A–C) patient.

Nat. Rev. Neurol. doi: /nrneurol

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Figure 1 MRI at presentation The axial diffusion-weighted image (A) showed restricted diffusion throughout the cortical ribbon of the right hemisphere.

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MR scans of brain and spine: (A) sagittal T2 image showing signal change in the posterior spinal cord between C3 and T6. MR scans of brain and spine: (A)

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Presentation transcript:

Figure 1. Prebiopsy and postbiopsy MRI Prebiopsy and postbiopsy MRI (A) Immediately prebiopsy, axial brain MRI shows a non–contrast enhancing T1 hyperintensity in the right basal ganglia juxtaposed to the external capsule (arrows) with modest mass effect; these changes were not evident in a prior MRI study. Corresponding fluid-attenuated inversion recovery (FLAIR) and diffusion-weighted imaging (DWI) sequences were essentially unremarkable. (B) Postbiopsy axial brain MRI reveals resolution of the right basal ganglia T1 hyperintensity (arrow), with biopsy artefact (arrow head), with corresponding FLAIR images remaining otherwise unremarkable aside from ongoing modest mass effect (arrow), while the DWI series displays subtle cerebral cortical signal increase in the posterior cingulate gyri (right more than left) and right posterior temporal region (arrow). Boaz Kim et al. Neurol Neuroimmunol Neuroinflamm 2016;3:e248 © 2016 American Academy of Neurology