This Month in Gastroenterology Jan Tack, John M. Carethers  Gastroenterology  Volume 139, Issue 5, Pages 1429-1432 (November 2010) DOI: 10.1053/j.gastro.2010.09.013 Copyright © 2010 AGA Institute Terms and Conditions

Figure 1 Effect of treatment on endoscopic signs. Endoscopic pictures taken from 3 patients with histologically confirmed active eosinophilic esophagitis at baseline and after successful treatment with budesonide achieving histologic remission, demonstrating reversibility of white exudates and red furrows, but persistence of corrugated rings. Gastroenterology 2010 139, 1429-1432DOI: (10.1053/j.gastro.2010.09.013) Copyright © 2010 AGA Institute Terms and Conditions

Figure 2 ORs and 95% CIs of risk factors for fecal incontinence among women in Olmsted County, Minnesota. Gastroenterology 2010 139, 1429-1432DOI: (10.1053/j.gastro.2010.09.013) Copyright © 2010 AGA Institute Terms and Conditions

Figure 3 Gene expression profiles of jejunal mucosa of Dicer1 mutants and controls. Differentially expressed genes were sorted into pathways, and the differentially activated pathways combined into functional groups. Immune pathways made up one third of the differentially expressed genes. Gastroenterology 2010 139, 1429-1432DOI: (10.1053/j.gastro.2010.09.013) Copyright © 2010 AGA Institute Terms and Conditions

Figure 4 Sera from BA infants at the time of diagnosis (left panel, IgM), BA children >1 year of age with their native liver (right panel, IgG), and other disease age-matched controls were tested for anti-enolase antibodies by ELISA. Each dot represents the average anti-enolase antibody level from a single patient; the bar is the mean value of all subjects within the group. Gastroenterology 2010 139, 1429-1432DOI: (10.1053/j.gastro.2010.09.013) Copyright © 2010 AGA Institute Terms and Conditions