Clearance of a fluconazole-resistant Candida albicans strain after switching antifungal therapy and initiation of triple therapy for HIV infection M.

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Clearance of a fluconazole-resistant Candida albicans strain after switching antifungal therapy and initiation of triple therapy for HIV infection M. Ruhnke, A. Adler, F.-M. Muller Clinical Microbiology and Infection Volume 6, Issue 4, Pages 220-223 (April 2000) DOI: 10.1046/j.1469-0691.2000.00041-1.x Copyright © 2000 European Society of Clinical Infectious Diseases Terms and Conditions

Figure 1 (a) Profiles of AP-PCR fingerprints are presented for 11 isolates of C. albicans from biopsies obtained from the esophagus and stomach of a single patient. Esophagus: lane 1, 9 May 1996; lane 3, 4 July 1996; lane 4, 23 July 1996; lane 6, 16 August 1996; lane 7, 14 November 1996; lane 8, 3 January 1997; lane 10, 7 February 1997). Stomach: lane 2, 9 May 1996; lane 5, 16 August 1996; lane 9, 3 March 1997; lane 11, 7 February 1997. Samples were amplified by the primer RP02 [5]. Genotypes presented in lanes 1-6 and 10 (genotype A) were considered to be identical, as well as those in lanes 8-9 and 11 (genotype B1). The genotype in lane 7 was similar to genotype B1 but differed in one band and was considered to be subtype B2. The molecular size marker DNA VI (Boehringer-Ingelheim, Ingelheim, Germany) is presented to the left of the gel (M). kb, kilobase. (b): Profiles of AP-PCR fingerprints are presented for 13 isolates of C. albicans from oral mouthwashes obtained from a single patient: lane 1, 19 April 1996; lane 2, 9 May 1996; lane 3, 17 July 1996; lane 4, 16 August 1996; lane 5, 14 November 1996; lane 6, 29 November 1996; lane 7, 13 December 1996; lane 8, 3 January 1997; lane 9, 7 February 1997; lane 10, 6 August 1997; lane 11, 27 August 1997; lane 12, 26 May 1997; lane 13, 12 June 1998. Samples were amplified by the primer RP02 [5]. Genotypes presented in lanes 1 and 7 were considered to be identical, as well as those in lanes 2-4, 6 and 8-13. The genotype in lane 5 was similar to genotype B1 but differed in one band and was considered to be subtype B2. kb, kilobase. Clinical Microbiology and Infection 2000 6, 220-223DOI: (10.1046/j.1469-0691.2000.00041-1.x) Copyright © 2000 European Society of Clinical Infectious Diseases Terms and Conditions