Volume 132, Issue 2, Pages (February 2007)

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Volume 132, Issue 2, Pages 826-827 (February 2007) Correction    Gastroenterology  Volume 132, Issue 2, Pages 826-827 (February 2007) DOI: 10.1053/j.gastro.2007.01.023 Copyright © 2007 AGA Institute Terms and Conditions

Figure 1 In situ phenotypic characterization of APCs in normal duodenal mucosa. Paired immunofluorescence staining for markers as indicated on cryosections of mucosal tissue. (A) Most HLA-DQ2+ cells co-express CD68+, but (B) a minor population express CD11c+ (arrowheads). (C) Virtually no cells co-express CD68 and CD11c. Note dendritic morphology of CD11c+ cells (arrowhead). (D) A subset of CD11c+ cells co-express CD1c (arrowheads). (E) Most macrophages express DC-SIGN. Surface epithelium is visualized in A–C (autofluorescence at excitation wave length of 650 nm; blue color). Gastroenterology 2007 132, 826-827DOI: (10.1053/j.gastro.2007.01.023) Copyright © 2007 AGA Institute Terms and Conditions

Figure 2 In situ phenotypic characterization of APCs in the celiac lesion. Paired immunofluorescence staining for markers as indicated on cryosections of duodenal mucosa from active celiac lesion. (A) Note an apparent increase in the density of CD11c+ cells compared with that in Figure 1B–D. (B) Some CD14+ monocytes do not express CD68 (arrowhead), indicating DC differentiation. (C) Some DC-SIGN+ cells express CD16 (arrowhead). (D) CD86 is expressed preferentially on CD11c+ DCs in the celiac lesion (arrowhead). Crypt epithelium is indicated with an asterisk (D). The basement membrane of surface epithelium is indicated by a broken line (A and D). Gastroenterology 2007 132, 826-827DOI: (10.1053/j.gastro.2007.01.023) Copyright © 2007 AGA Institute Terms and Conditions