Desmoid Tumors in a Dutch Cohort of Patients With Familial Adenomatous Polyposis  Marry H. Nieuwenhuis, Wouter De Vos Tot Nederveen Cappel, Akke Botma, Fokko M. Nagengast, Jan H. Kleibeuker, Elisabeth M.H. Mathus–Vliegen, Evelien Dekker, Jan Dees, Juul Wijnen, Hans F.A. Vasen  Clinical Gastroenterology and Hepatology  Volume 6, Issue 2, Pages 215-219 (February 2008) DOI: 10.1016/j.cgh.2007.11.011 Copyright © 2008 AGA Institute Terms and Conditions

Figure 1 Cumulative risk of desmoid development. Clinical Gastroenterology and Hepatology 2008 6, 215-219DOI: (10.1016/j.cgh.2007.11.011) Copyright © 2008 AGA Institute Terms and Conditions

Figure 2 Representation of the APC gene and the distribution of mutations of families with a desmoid tumor. The number of dots corresponds with the frequency of mutations at one particular site. Clinical Gastroenterology and Hepatology 2008 6, 215-219DOI: (10.1016/j.cgh.2007.11.011) Copyright © 2008 AGA Institute Terms and Conditions

Figure 3 Cumulative risk of desmoid development for patients who have minimal 1 family member with a desmoid tumor (grey), compared with patients without desmoids (black) in the family history. Clinical Gastroenterology and Hepatology 2008 6, 215-219DOI: (10.1016/j.cgh.2007.11.011) Copyright © 2008 AGA Institute Terms and Conditions