Nat. Rev. Nephrol. doi: /nrneph

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Nat. Rev. Nephrol. doi: /nrneph

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Figure 5 A layered approach to the follow-up of patients with acute kidney disease (AKD) Figure 5 | A layered approach to the follow-up of patients with.

Figure 4 Interplay between acute kidney injury (AKI),

Figure 6 Effects of adiponectin on podocyte function

Figure 1 Pre-eclampsia prevents VEGF signalling in endothelial cells

Figure 4 The theoretical contribution of genetic and

Figure 3 Energy metabolism regulation, cardiovascular and bone disease in CKD Figure 3 | Energy metabolism regulation, cardiovascular and bone disease.

Figure 4 Expression of coagulation protease receptors in renal cells

Figure 3 Angiotensin signalling in adiporenal crosstalk

Figure 4 Interactions between adipose, the microbiome and kidney

Nat. Rev. Nephrol. doi: /nrneph

Figure 1 Mechanisms of kidney injury in the setting of obesity

Nat. Rev. Nephrol. doi: /nrneph

Figure 1 Role of the kidney in glucose homeostasis

Nat. Rev. Nephrol. doi: /nrneph

Nat. Rev. Nephrol. doi: /nrneph

Figure 1 The structure of the glycocalyx

Figure 6 The bioavailability of phosphate differs according to the protein source Figure 6 | The bioavailability of phosphate differs according to the.

Figure 7 The efficacy of phosphate-binder therapy

Figure 3 Putative actions of glucagon-like peptide 1 (GLP-1)

Nat. Rev. Nephrol. doi: /nrneph

Figure 2 The network of chronic diseases and their mutual influences

Figure 2 Three distinct mechanisms of activation of

Figure 1 The burden of chronic kidney disease (CKD)

Figure 3 Societal costs for the care of patients with chronic kidney disease in the UK Figure 3 | Societal costs for the care of patients with chronic.

Figure 2 Podocyte dysfunction is a common feature of renal injury

Nat. Rev. Nephrol. doi: /nrneph

Figure 1 Acute kidney injury and chronic kidney disease

Figure 1 Innate immune responses in atherosclerosis

Figure 4 The gut–kidney axis, inflammation and cardiovascular disease in CKD Figure 4 | The gut–kidney axis, inflammation and cardiovascular disease in.

Figure 2 The continuum of acute kidney injury (AKI),

Figure 4 Model of changes in the serum levels

Nat. Rev. Nephrol. doi: /nrneph

Figure 5 Consequences of CKD on lipid metabolism

Figure 2 Mechanism of glycocalyx degradation

Nat. Rev. Nephrol. doi: /nrneph

Figure 3 Differentiation and functional control of T-cell subsets

Nat. Rev. Cardiol. doi: /nrcardio

Figure 3 Cascade of events leading from AKI to ALI

Figure 4 The molecular configuration of the CD20 molecule

Nat. Rev. Nephrol. doi: /nrneph

Nat. Rev. Nephrol. doi: /nrneph

Nat. Rev. Nephrol. doi: /nrneph

Figure 4 Cascade system activation on a biomaterial surface

Nat. Rev. Nephrol. doi: /nrneph

Figure 3 Loss of the glycocalyx leads to podocyte and kidney injury

Nat. Rev. Nephrol. doi: /nrneph

Figure 3 LDL autoimmunity in atherosclerosis

Nat. Rev. Nephrol. doi: /nrneph

Nat. Rev. Nephrol. doi: /nrneph

Nat. Rev. Nephrol. doi: /nrneph

Figure 4 Podocyte–endothelial cross talk and activation of heparanase

Figure 3 Biologics that attenuate effector responses in the kidney

Nat. Rev. Nephrol. doi: /nrneph

Figure 3 Serum phosphate level is associated with

Nat. Rev. Nephrol. doi: /nrneph

Nat. Rev. Nephrol. doi: /nrneph

Figure 2 Adaptive immunity in atherosclerosis

Figure 1 Mechanistic classification of lncRNAs

Nat. Rev. Nephrol. doi: /nrneph

Figure 2 Biologics that target CD4+ T helper (TH)-cell subsets

End-stage renal disease, atherosclerosis, and cardiovascular mortality: Is C-reactive protein the missing link? Mustafa Arici, John Walls Kidney International

Figure 1 Patient, facility, health-care system and industry factors

Figure 3 Preventive strategies for CSA-AKI

Figure 4 Intracellular distribution and

Figure 1 Worldwide distribution of disease burden attributable to environmental risks in 2012 Figure 1 | Worldwide distribution of disease burden attributable.

primary Sjögren syndrome

End-stage renal disease, atherosclerosis, and cardiovascular mortality: Is C-reactive protein the missing link? Mustafa Arici, John Walls Kidney International

Nat. Rev. Nephrol. doi: /nrneph

Figure 1 Relationships between genetic variants, quantitative traits and diseases Figure 1 | Relationships between genetic variants, quantitative traits.

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Nat. Rev. Nephrol. doi:10.1038/nrneph.2015.162 Figure 4 Development of atherosclerotic lesions after loss of the glycocalyx Figure 4 | Development of atherosclerotic lesions after loss of the glycocalyx. a | Normally, the endothelium (blue) is covered by the glycocalyx, which prevents the passage of proteins and lipoproteins into the subendothelial space. b | Following loss of the glycocalyx, proteins and lipoproteins can enter the subendothelial space. The associated recruitment of inflammatory cells drives formation of atherosclerotic lesions. Rabelink, T. J. & de Zeeuw, D. (2015) The glycocalyx—linking albuminuria with renal and cardiovascular disease Nat. Rev. Nephrol. doi:10.1038/nrneph.2015.162