Autophagy, Microbial Sensing, Endoplasmic Reticulum Stress, and Epithelial Function in Inflammatory Bowel Disease Arthur Kaser, Richard S. Blumberg Gastroenterology Volume 140, Issue 6, Pages 1738-1747.e2 (May 2011) DOI: 10.1053/j.gastro.2011.02.048 Copyright © 2011 AGA Institute Terms and Conditions
Figure 1 Molecules involved in autophagy induction. Gastroenterology 2011 140, 1738-1747.e2DOI: (10.1053/j.gastro.2011.02.048) Copyright © 2011 AGA Institute Terms and Conditions
Figure 2 Experimental measures to determine induction of autophagy. Gastroenterology 2011 140, 1738-1747.e2DOI: (10.1053/j.gastro.2011.02.048) Copyright © 2011 AGA Institute Terms and Conditions
Figure 3 Signaling pathways induced by NOD2 activation. Gastroenterology 2011 140, 1738-1747.e2DOI: (10.1053/j.gastro.2011.02.048) Copyright © 2011 AGA Institute Terms and Conditions
Figure 4 Proximal effectors of the unfolded protein response. Gastroenterology 2011 140, 1738-1747.e2DOI: (10.1053/j.gastro.2011.02.048) Copyright © 2011 AGA Institute Terms and Conditions
Figure 5 Relationship between the unfolded protein response, in response to endoplasmic reticulum stress, autophagy, and intracellular bacterial and viral sensing. Gastroenterology 2011 140, 1738-1747.e2DOI: (10.1053/j.gastro.2011.02.048) Copyright © 2011 AGA Institute Terms and Conditions
Gastroenterology 2011 140, 1738-1747. e2DOI: (10. 1053/j. gastro. 2011 Copyright © 2011 AGA Institute Terms and Conditions
Gastroenterology 2011 140, 1738-1747. e2DOI: (10. 1053/j. gastro. 2011 Copyright © 2011 AGA Institute Terms and Conditions