Acquired type 2A von Willebrand syndrome caused by aortic valve disease corrects during valve surgery C. Solomon, U. Budde, S. Schneppenheim, E. Czaja,

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Acquired type 2A von Willebrand syndrome caused by aortic valve disease corrects during valve surgery C. Solomon, U. Budde, S. Schneppenheim, E. Czaja, C. Hagl, H. Schoechl, M. von Depka, N. Rahe-Meyer British Journal of Anaesthesia Volume 106, Issue 4, Pages 494-500 (April 2011) DOI: 10.1093/bja/aeq413 Copyright © 2011 The Author(s) Terms and Conditions

Fig 1 Densitometric (a) and gel electrophoresis (b) analysis of vWF HMWM in one subject with AV stenosis. (a) Analyses performed (1) before surgery (large multimers 19.6%, intermediate multimers 37.8%, small multimers 42.6%); (2) immediately after weaning from CPB (large multimers 36.6%, intermediate multimers 31.6%, small multimers 31.8%); and (3) on the first postoperative day (large multimers 32.7%, intermediate multimers 37.7%, and small multimers 29.6%). (b) Analyses performed (1) before surgery; (2) immediately after weaning from CPB; and (3) on the first postoperative day; (4) in plasma pooled from 30 normal blood donors. British Journal of Anaesthesia 2011 106, 494-500DOI: (10.1093/bja/aeq413) Copyright © 2011 The Author(s) Terms and Conditions

Fig 2 Perioperative changes in vWF antigen and vWF collagen-binding capacity in subjects undergoing AV surgery. Measurements performed before operation (time point 1), at the end of the CPB (time point 2), and on the first postoperative day (time point 3). VWF:Ag, von Willebrand factor antigen; VWF:CB, von Willebrand factor collagen-binding capacity. British Journal of Anaesthesia 2011 106, 494-500DOI: (10.1093/bja/aeq413) Copyright © 2011 The Author(s) Terms and Conditions