Volume 86, Issue 6, Pages (December 2014)

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Presentation transcript:

Volume 86, Issue 6, Pages 1074-1076 (December 2014) Factors influencing clinical outcome in patients with primary hyperoxaluria type 1 Sonia Fargue Kidney International Volume 86, Issue 6, Pages 1074-1076 (December 2014) DOI: 10.1038/ki.2014.280 Copyright © 2014 International Society of Nephrology Terms and Conditions

Figure 1 Factors potentially influencing the metabolism of oxalate and renal outcome in patients with primary hyperoxaluria type 1. AGT, alanine glyoxylate aminotransferase; B6, vitamin B6 (all vitamers); Ca, calcium; CaOx, calcium oxalate; G170R, p.Gly170Arg mutation; GO, glycolate oxidase; GR, glyoxylate reductase; HOGA, 4-hydroxy-2-oxoglutarate aldolase; mis, AGT missense mutation (other than G170R); null, AGT null mutation; PLP, pyridoxal-5′-phosphate; SLC26, SLC26 family of anion transporters. Plus and minus signs indicate favorable and unfavorable influence on primary hyperoxaluria (PH) evolution, respectively. Solid arrows indicate known influences in the metabolism of glyoxylate; dashed arrows, potential influences; and question marks, unknown factors. Endogenous factors have been shown to play the main role in the pathophysiology of PH1, PH2, and PH3. Kidney International 2014 86, 1074-1076DOI: (10.1038/ki.2014.280) Copyright © 2014 International Society of Nephrology Terms and Conditions