IgA Autoimmune Disorders: Development of a Passive Transfer Mouse Model  John J. Zone, C. Anthony Egan, Ted B. Taylor  Journal of Investigative Dermatology Symposium Proceedings  Volume 9, Issue 1, Pages 47-51 (January 2004) DOI: 10.1111/j.1087-0024.2004.00840.x Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions

Figure 1 Diagrammatic representation of the transmembrane hemidesmosomal protein BP180 (BPAg2). The primary site of antigenic reactivity for bullous pemphigoid (MCW1) is different from that for linear IgA bullous dermatosis (LABD97). Journal of Investigative Dermatology Symposium Proceedings 2004 9, 47-51DOI: (10.1111/j.1087-0024.2004.00840.x) Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions

Figure 2 Infiltration of dermal papillae with neutrophils and mononuclear cells after administration of IgA BMZ antibodies (H&E 40 X). Journal of Investigative Dermatology Symposium Proceedings 2004 9, 47-51DOI: (10.1111/j.1087-0024.2004.00840.x) Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions

Figure 3 BMZ vesicle with neutrophil infiltration after administration of IgA BMZ antibodies (H&E 40X). Journal of Investigative Dermatology Symposium Proceedings 2004 9, 47-51DOI: (10.1111/j.1087-0024.2004.00840.x) Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions