Focus on nasopharyngeal carcinoma

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Focus on nasopharyngeal carcinoma Kwok Wai Lo, Ka Fai To, Dolly P Huang Cancer Cell Volume 5, Issue 5, Pages 423-428 (May 2004) DOI: 10.1016/S1535-6108(04)00119-9

Figure 1 EBV latent infection in nasopharyngeal carcinoma A and B: Typical nasopharyngeal undifferentiated carcinoma (WHO type III). A: The carcinoma cells exhibit prominent nucleoli and arrange in syncytial clusters. Dense infiltration of lymphocytes and plasma cells is evident (hematoxylin and eosin stain [H&E], 400×). B: In situ hybridization (ISH) for EBV RNA (EBER) reveals distinct nuclear positivity, indicative of latent infection in carcinoma cells (400×). C and D: Squamous cell carcinoma (SCC, WHO type I) from endemic region. C: SCC with intercellular bridges and keratinization is seen (H&E, 400×). D: ISH-EBV is positive (400×). E and F: SCC (WHO type I) from nonendemic region. E: SCC with intercellular bridges, keratinization, and keratin pearl are noted (H&E, 400×). F: ISH-EBV is negative (400×). Cancer Cell 2004 5, 423-428DOI: (10.1016/S1535-6108(04)00119-9)

Figure 2 Multistep carcinogenesis of nasopharyngeal carcinoma Hematoxylin and eosin staining and EBER in situ hybridization of the dysplastic lesions and invasive carcinoma are shown in the upper and lower panels, respectively. Cancer Cell 2004 5, 423-428DOI: (10.1016/S1535-6108(04)00119-9)