Targets to retard the progression of diabetic nephropathy Mark E. Cooper, Karin Jandeleit-Dahm, Merlin C. Thomas Kidney International Volume 68, Issue 4, Pages 1439-1445 (October 2005) DOI: 10.1111/j.1523-1755.2005.00555.x Copyright © 2005 International Society of Nephrology Terms and Conditions
Figure 1 Glomerular hypertrophy and mesangial expansion in diabetic c57 and diabetic apolipoprotein E knockout mice. (A) c57. (B) Diabetic c57. (C) Apolipoprotein E knockout control. (D) Diabetic apolipoprotein E knockout [periodic acid-Schiff (PAS) staining ×400]. Kidney International 2005 68, 1439-1445DOI: (10.1111/j.1523-1755.2005.00555.x) Copyright © 2005 International Society of Nephrology Terms and Conditions
Figure 2 The prevalence of functional erythropoietin (EPO) deficiency (<31 IU/mL) and reduced iron availability [transferrin saturation (TSAT) <16%] in patients with type 2 diabetes and anemia, stratified according to the renal function. GFR is glomerular filtration rate. Kidney International 2005 68, 1439-1445DOI: (10.1111/j.1523-1755.2005.00555.x) Copyright © 2005 International Society of Nephrology Terms and Conditions