Deep hypothermic circulatory arrest: II

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Presentation transcript:

Deep hypothermic circulatory arrest: II Deep hypothermic circulatory arrest: II. Changes in electroencephalogram and evoked potentials during rewarming Mark M Stecker, MD, PhD, Albert T Cheung, MD, Alberto Pochettino, MD, Glenn P Kent, BS, Terry Patterson, PhD, Stuart J Weiss, MD, PhD, Joseph E Bavaria, MD The Annals of Thoracic Surgery Volume 71, Issue 1, Pages 22-28 (January 2001) DOI: 10.1016/S0003-4975(00)02021-X

Fig 1 (A) Dependence of the N20-P22 amplitude on temperature during cooling. The line represents a sigmoidal curve fit to data obtained from 31 neurologically normal patients. The temperature at which various electroencephalogram (EEG) and evoked potential events are illustrated on the graph as are their standard deviations. (B) Dependence of the N20-P22 amplitude on temperature during rewarming after circulatory arrest. The line represents a sigmoidal curve fit to data obtained from 32 neurologically normal patients. The temperature at which various EEG and evoked potential events are illustrated on the graph as are their standard deviations. The difference in sequence of electrophysiologic events in these two figures is clear. Only 31 and 32 patients, respectively, were randomly chosen out of the 69 possible neurologically normal patients to provide an illustration of the changes in amplitudes as a function of temperature. The Annals of Thoracic Surgery 2001 71, 22-28DOI: (10.1016/S0003-4975(00)02021-X)

Fig. 2 Receiver operator characteristic curve demonstrating the sensitivity and specificity of predicting postoperative neurologic impairment based on NTRCont and NTRN20 using a logistic regression analysis (Table 4). The Annals of Thoracic Surgery 2001 71, 22-28DOI: (10.1016/S0003-4975(00)02021-X)