Figure 4 Local species pools that contribute to the

Slides:



Advertisements
Similar presentations
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Advertisements

Figure 3 Low-grade inflammation in FGID
Figure 4 The gut microbiota directly influences T-cell differentiation
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Imaging of a depressed intramucosal carcinoma
Figure 4 Activation of clopidogrel via cytochrome P450
Figure 5 Therapeutic paradigms for interfering with the brain–gut axis
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Gut microorganisms at the intersection of several diseases
Figure 5 Lipid droplet consumption
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Host factors influencing gut bacterial growth
Figure 2 The microbiome–gut–brain axis
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Biosimilar development process
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 3 The 'leaky gut' hypothesis
Figure 2 Effect of PPIs on gastric physiology
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 4 Giant lipid droplet formation
Figure 1 Schematic outlining the results of Buffington et al.
Figure 6 Combination therapy for HCC
Figure 2 Modelling the effect of HCV treatment on reinfection in people who inject drugs Figure 2 | Modelling the effect of HCV treatment on reinfection.
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Host range of hepatitis E virus
Figure 2 Switching of biologic agents and biosimilars
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 3 Example wireless motility recording
to the liver and promote patient-derived xenograft tumour growth
Figure 7 Example colonic high-resolution manometry
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Environmental factors contributing to IBD pathogenesis
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 3 Clinical algorithms in the management of NASH and diabetes mellitus Figure 3 | Clinical algorithms in the management of NASH and diabetes mellitus.
Figure 1 Median coverage and distribution by
in the UK (1961–2012), France (1961–2014) and Italy (1961–2010)
Figure 5 Representative barium defecography images
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 6 Possible therapeutic targets to decrease hepatic steatosis
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 5 High-resolution manometry studies performed
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 4 The gut–liver relationship in PSC
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Brain–gut axis Brain–gut axis. Schematic of the brain–gut axis, including inputs from the gut microbiota, the ENS, the immune system and the external.
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 3 Optical coherence tomography images of specialized intestinal
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 6 Assessment of colonic transit time with radiopaque markers
Figure 5 Systems biological model of IBS
Figure 1 Mucosal immune networks
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 2 Lifelong influences on the gut microbiome from
Figure 1 Colonic inflammation in IBD and link to the gut microbiota
Figure 2 Classifications and appearance of CCAs
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 2 Enhanced imaging techniques and autofluorescence endoscopy for Barrett oesophagus Figure 2 | Enhanced imaging techniques and autofluorescence.
Figure 3 Endomicroscopy for optical intestinal biopsy
Presentation transcript:

Figure 4 Local species pools that contribute to the regional pool of microorganisms available for colonization of the infant gut Figure 4 | Local species pools that contribute to the regional pool of microorganisms available for colonization of the infant gut. Infants are colonized by microorganisms from host-associated communities, environmental communities that are not host adapted, and unknown microbial sources. The thickness of the arrows denotes the hypothesized relative contributions of microorganisms from different sources that disperse to and stably colonize the local community (infant gastrointestinal tract). Sprockett, D. et al. (2018) Role of priority effects in the early-life assembly of the gut microbiota Nat. Rev. Gastroenterol. Hepatol. doi:10.1038/nrgastro.2017.173