Volume 69, Issue 1, Pages 262-264 (July 2018) Erratum to “A new HDV mouse model identifies mitochondrial antiviral signaling protein (MAVS) as a key player in IFN-β induction” [J Hepatol 67 (2017) 669–679]  Lester Suárez-Amarán, Carla Usai, Marianna Di Scala, Cristina Godoy, Yi Ni, Mirja Hommel, Laura Palomo, Víctor Segura, Cristina Olagüe, Africa Vales, Alicia Ruiz-Ripa, Maria Buti, Eduardo Salido, Jesús Prieto, Stephan Urban, Francisco Rodríguez-Frias, Rafael Aldabe, Gloria González-Aseguinolaza  Journal of Hepatology  Volume 69, Issue 1, Pages 262-264 (July 2018) DOI: 10.1016/j.jhep.2018.04.003 Copyright © 2018 Terms and Conditions

Fig. 1 Infection of C57BL/6 mice with AAV-HBV/HDV gives rise to the production of circulating HDV infectious particles and a detectable humoral immune response against viral antigens, while anti-HBsAg and anti-HDAg humoral response is induced. (This figure appears in colour on the web.) Journal of Hepatology 2018 69, 262-264DOI: (10.1016/j.jhep.2018.04.003) Copyright © 2018 Terms and Conditions

Fig. 2 Infection of RAGB6 mice with AAV-HBV/HDV gives rise to the sustained production of circulating HDV infectious particles and HBV particles. (This figure appears in colour on the web.) Journal of Hepatology 2018 69, 262-264DOI: (10.1016/j.jhep.2018.04.003) Copyright © 2018 Terms and Conditions

Fig. 8 AAV-HDV infection induces an innate immune response that is partially dependent on MAVS. (This figure appears in colour on the web.) Journal of Hepatology 2018 69, 262-264DOI: (10.1016/j.jhep.2018.04.003) Copyright © 2018 Terms and Conditions