Figure 5 Interaction between infarction and amyloid deposition

Slides:

Advertisements

Similar presentations

Table 3 Limb-girdle muscular dystrophy standardized data collection form and HPO mapping Thompson, R. & Straub, V. et al. (2016) Limb-girdle muscular dystrophies.

Advertisements

Nat. Rev. Neurol. doi: /nrneurol

Figure Pedigrees of the SCA42 families identified in this study

Nat. Rev. Neurol. doi: /nrneurol

Figure 3 Life expectancy at birth in all countries included

Nat. Rev. Neurol. doi: /nrneurol

Figure 2 The US Centers for Disease Control and

Figure 1 Carotid artery inflammation detected using 18F-FDG-PET

Figure 2 Overview of risk factors for Parkinson disease dementia

Nat. Rev. Neurol. doi: /nrneurol

Figure 3 Proportion of patients for whom NEDA

Figure 2 Use of a brain–computer interface in severe chronic stroke

Figure 3 Associations of [11C](R)-PK11195 binding to disability and diffusion tensor imaging (DTI) changes in patients with MS Associations of [11C](R)-PK11195.

Figure 8 4D magnetic resonance imaging patterns

Nat. Rev. Neurol. doi: /nrneurol

Figure 6 Pharmacodynamic profile of switching between clopidogrel and ticagrelor therapy: results from the RESPOND study Figure 6| Pharmacodynamic profile.

Figure 3 LGI1 and Caspr2 immunohistochemistry on rat brain

Nat. Rev. Urol. doi: /nrurol

Figure Sural nerve electron microscopy

Figure 2 Algorithm for immunosuppressive treatment

Nat. Rev. Neurol. doi: /nrneurol

Figure 1 Principles of therapeutic ultrasound

Nat. Rev. Neurol. doi: /nrneurol

Figure 3 Example of venous narrowing

Figure 1 Clinical correlates of neurodegeneration in MS

Figure 3 Genetic pleiotropy in ALS

Nat. Rev. Neurol. doi: /nrneurol

Figure 1 Evolution of multiple sclerosis

Figure 2 Haemorrhagic stroke prevalence and mortality

Nat. Rev. Cardiol. doi: /nrcardio

Nat. Rev. Neurol. doi: /nrneurol

Figure 4 Detection of EBER+ cells in MS and control brains by in situ hybridization Detection of EBER+ cells in MS and control brains by in situ hybridization.

Figure 5 Role of VEGF in stroke

Figure 3 Stroke-related deaths and DALYs by country development status

Figure 4 11C-PK11195-PET scans showing the evolution of neuroinflammation in a patient after stroke Figure 4 | 11C-PK11195-PET scans showing the evolution.

Nat. Rev. Neurol. doi: /nrneurol

Figure 2 Carotid artery inflammation detected using 68Ga-DOTATATE-PET

Figure 6 Combining population-wide and high-risk strategies

Figure 1 Ischaemic stroke prevalence and mortality

Figure 3 Optical coherence tomography images of specialized intestinal

Nat. Rev. Cardiol. doi: /nrcardio

Figure 3 Potential interplay between physiological

Figure 1 The VEGF family of growth factors

Figure 1 Tau PET images in patients with Alzheimer disease

Figure 5 Neurite structure is not disrupted by the lack of neurofilament light (NEFL)‏ Neurite structure is not disrupted by the lack of neurofilament.

Figure 2 Overview of the patient's history and immunofluorescence pattern of patient CSF IgG Overview of the patient's history and immunofluorescence pattern.

Figure 2 Linkage analysis of chromosome 19

Figure 1 Timing of the effects of deep brain stimulation

Figure 1 A large number of genes are potentially associated with CIPN

Nat. Rev. Neurol. doi: /nrneurol

Figure 1 Illustration of white matter– and lesion-associated regions of interest (ROIs)‏ Illustration of white matter– and lesion-associated regions of.

Figure 1 Family pedigree and DNA sequencing results

Figure 1 [18F]florbetapir standardized uptake value ratio analytical method [18F]florbetapir standardized uptake value ratio analytical method Flowchart.

Nat. Rev. Cardiol. doi: /nrcardio

Nat. Rev. Neurol. doi: /nrneurol

Figure 2 A case of early-onset PSD

Figure 1 Histamine flare in patients and controls

Figure 2 Global tau-PET distribution in familial prion disease mirrors the distribution seen in Alzheimer disease Global tau-PET distribution in familial.

Figure 1 Stacked bar chart depicts the proportion of patients with diffusion-weighted imaging (DWI)+ and DWI− scans categorized by index event type TIA.

Figure 2 C5B3 prevented AQP4-IgG–mediated CDC without affecting AQP4-IgG binding to AQP4 C5B3 prevented AQP4-IgG–mediated CDC without affecting AQP4-IgG.

Figure 2 Network connections of the hypothalamus

Yian Gu et al. Neurol Neuroimmunol Neuroinflamm 2019;6:e521

Ingo Kleiter et al. Neurol Neuroimmunol Neuroinflamm 2018;5:e504

Gitanjali Das et al. Neurol Neuroimmunol Neuroinflamm 2018;5:e453

Figure 2 Pedigrees of families and segregation analysis of variants c

Figure 2 MRIs (cases 2 and 3)‏

Figure 3 C5B3 blocked MAC formation

Figure 2 Aβ-PET scans obtained using different tracers

Figure 2 Region of interest (ROI)-specific [11C](R)-PK11195 binding in patients with relapsing-remitting MS (RRMS), patients with secondary progressive.

Figure 2 Correlations of subcortical gray matter SUVRs with the EDSS score, T25FW, and BPV in MS Correlations of subcortical gray matter SUVRs with the.

Presentation transcript:

Figure 5 Interaction between infarction and amyloid deposition Figure 5 | Interaction between infarction and amyloid deposition. 11C-Pittsburgh B compound (PiB) binding is increased (higher distribution volume ratio, DVR) in the peri-infarct region and within the infarct in the affected hemisphere (red arrow) compared with the corresponding region in the contralateral hemisphere (green arrow). Scale bar: DVR. Reproduced with permission from Wolters Kluwer Health © Ly, J. V. et al. Stroke 43, 1341–1346 (2012). Reproduced with permission from Wolters Kluwer Health © Ly, J. V. et al. Stroke 43, 1341–1346 (2012). Evans, N. R. et al. (2017) PET imaging of the neurovascular interface in cerebrovascular disease Nat. Rev. Neurol. doi:10.1038/nrneurol.2017.129