Coexistent aortic dissection and hepatocellular carcinoma with elevated plasma transforming growth factor β level: Possible roles of fibrillin 1 and transforming.

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Coexistent aortic dissection and hepatocellular carcinoma with elevated plasma transforming growth factor β level: Possible roles of fibrillin 1 and transforming growth factor β Hisato Takagi, MD, PhD, Hideaki Manabe, MD, Seishiro Sekino, MD, Takayoshi Kato, MD, Yukihiro Matsuno, MD, PhD, Takuya Umemoto, MD, PhD The Journal of Thoracic and Cardiovascular Surgery Volume 129, Issue 2, Pages 462-463 (February 2005) DOI: 10.1016/j.jtcvs.2004.06.033 Copyright © 2005 The American Association for Thoracic Surgery Terms and Conditions

Figure 1 Computed tomographic scanning disclosed both Stanford type A acute aortic dissection (A) and hepatic tumor, relatively hypodense in delayed phase (B). Postoperative computed tomographic scanning revealed hepatic lesion to be hyperdense in arterial phase (C). The Journal of Thoracic and Cardiovascular Surgery 2005 129, 462-463DOI: (10.1016/j.jtcvs.2004.06.033) Copyright © 2005 The American Association for Thoracic Surgery Terms and Conditions