Vitamin D3 levels alter amphetamine-related behaviors in mice.

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Anthony S. Rauhut 1,2 and André White 2, Department of Psychology 1 and Neuroscience Program 2, Dickinson College, Carlisle, PA References Discussion Abstract.
Representative 10-s EEG recordings from GAERS: during baseline (prior to injection of any drug) (i), following the i.c.v. injection of 4 μl of vehicle.
Prostaglandin E2 (PGE2) induces no significant alteration in circadian locomotor activity. Prostaglandin E2 (PGE2) induces no significant alteration in.
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The ERK phosphorylation inhibitor U0126 did not block the memory-enhancing effects of E2 in intact or GDX males. The ERK phosphorylation inhibitor U0126.
Active avoidance is impaired in aged mice in both multimodal and visual versions of the task. Active avoidance is impaired in aged mice in both multimodal.
Peptidergic nociceptor markers are reduced in DRG from Nav-Tsc2 mice.
Chemogenetic modulation of vlPAG glutamatergic neurons bidirectionally modulates nociceptive behaviors. Chemogenetic modulation of vlPAG glutamatergic.
Effects of E2 on levels of phospho-CREB in the DH of OVX females, sham males, and GDX males. Effects of E2 on levels of phospho-CREB in the DH of OVX females,
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Simultaneously recorded patterns of wheel running (RPM; black solid lines) and core body temperature (red broken lines) in three individual mice housed.
Exogenous CRP administration causes fasting hyperglycemia and hyperinsulinemia without altering body composition. Exogenous CRP administration causes fasting.
CNS administration of XPro1595 does not affect TMEV-induced acute seizure frequency and intensity. CNS administration of XPro1595 does not affect TMEV-induced.
Platelet-released serotonin contributes to hypothermia in mice undergoing HA-hIgG–dependent anaphylaxis. Platelet-released serotonin contributes to hypothermia.
Modulation of high γ activity in different brain regions is correlated with behavior. Modulation of high γ activity in different brain regions is correlated.
Expression of candidate neuronal IEGs in the brain of cycloheximide pretreated crickets 30 min after PTX injection. Expression of candidate neuronal IEGs.
Adult conditional gene deletion of PGC-1α.
Correlation of EEG- and behavioral-based estimates of pairwise face similarity. Correlation of EEG- and behavioral-based estimates of pairwise face similarity.
The complexity as well as the total dendritic length of ApoE-deficient adult-born granule cells are significantly reduced. The complexity as well as the.
The complexity and the total dendritic length of ApoE4 adult-born granule cells are significantly reduced. The complexity and the total dendritic length.
Characterization of non-DA firing.
Activation of glial cells.
Results of CFC training and testing.
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Loss of ATF3 function modestly reduces axonal regeneration following sciatic nerve crush. Loss of ATF3 function modestly reduces axonal regeneration following.
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BNST AVP cell ablations in iCre+ animals did not influence anxiety-like behavior. BNST AVP cell ablations in iCre+ animals did not influence anxiety-like.
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NRG1 activates cortical erbB4 receptors and increases excitation onto PV+ interneurons. NRG1 activates cortical erbB4 receptors and increases excitation.
Transgenic expression of mCREB or CREB produces opposite effects in the learned helplessness model of depression. Transgenic expression of mCREB or CREB.
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A, B, Startle magnitude and PPI after treatment with CNO
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MET-1 increases DRG neuron K+ currents.
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HFD increased the number of ERα-expressing cells.
FMRP S499 rephosphorylation kinetics after CX-4945 treatment and washout mirrors a known CK2 target. FMRP S499 rephosphorylation kinetics after CX-4945.
Spatial frequency preferences of contralateral responses is higher than ipsilateral responses in AAV-SynGCaMP6s-injected mice. Spatial frequency preferences.
Higher spatial frequency tuning of the contralateral responses also found in anesthetized animals. Higher spatial frequency tuning of the contralateral.
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Intra-amygdala MPEP is analgesic during bladder distention.
Grand-averaged ERPs across (A) left hemisphere electrodes (P5, P7, P9, PO3, PO7, and O1) and (B) right hemisphere electrodes (P6, P8, P10, PO4, PO8, and.
OGD treatment increased tonic current through an oxidative reaction.
Effects of CNO on spontaneous locomotion and amphetamine-induced hyperlocomotion. Effects of CNO on spontaneous locomotion and amphetamine-induced hyperlocomotion.
Experimental design. Experimental design. Animals were randomized to stimulation groups according to amphetamine rotation results (Amph-Rot) on week 10.
Both the MMP9 and MMP2 systems are activated by anti-Aβ antibodies, IVIg, and mouse IgG. Both the MMP9 and MMP2 systems are activated by anti-Aβ antibodies,
PNU enhances Ca2+ flux mediated by an engineered variant of the human α7 nAChR. PNU enhances Ca2+ flux mediated by an engineered variant.
Acute applications display only slight pharmacological differences among menthol stereoisomers. Acute applications display only slight pharmacological.
Simvastatin does not correct AGS in the Fmr1-/y mouse.
Effects of rapamycin, cycloheximide, and an MAP kinase inhibitor (U0126) on BDNF-mediated increase in Arc and CaMKII in acute hippocampal slices. Effects.
Presentation transcript:

Vitamin D3 levels alter amphetamine-related behaviors in mice. Vitamin D3 levels alter amphetamine-related behaviors in mice. A, Binned locomotor activity of HF and HF-D mice during habituation, saline injection (black arrow), and acute amphetamine treatment (red arrow). B, HF and HF-D cumulative activity for 20 min after amphetamine administration (n = 5, 5). C, D, Binned locomotor activity of Ch and Ch-D mice during habituation, saline injection (black arrow), and acute amphetamine treatment (red arrow; C), and Ch and Ch-D cumulative activity for 2 h after amphetamine (n = 4, 4; D). E, Binned locomotor activity in naïve mice pretreated with vehicle or calcitriol (n = 8, 8) 6-7 h prior to acute amphetamine treatment (red arrow). F, Cumulative activity of the pretreated mice over 2 h after amphetamine administration. Joseph R. Trinko et al. eneuro 2016;3:ENEURO.0122-15.2016 ©2016 by Society for Neuroscience