Treatment of Nephrotic Syndrome: Retrospection

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Presentation transcript:

Treatment of Nephrotic Syndrome: Retrospection Jerry Yee, MD Advances in Chronic Kidney Disease Volume 21, Issue 2, Pages 115-118 (March 2014) DOI: 10.1053/j.ackd.2014.01.012 Copyright © 2014 National Kidney Foundation, Inc. Terms and Conditions

Figure 1 Dyslipidemia of nephrotic syndrome. A glomerular disorder leads to excessive proteinuria with urinary protein losses. Urinary plasminogen losses induce ENaC sodium reabsorption, leading to edema and hypertension. Urinary losses of albumin, LCAT, and potentially other proteins lead to hepatic and extrahepatic biochemical alterations that produce elevations of CHOL and TG. Abbreviations: CHOL, cholesterol; ENaC, epithelial sodium channel; LCAT, lecithin cholesterol acyl transferase; TG, triglycerides. Diagram by Pablo Buitron de la Vega and Jerry Yee (iMindmap v. 6.0, ThinkBuzan, Cardiff). Advances in Chronic Kidney Disease 2014 21, 115-118DOI: (10.1053/j.ackd.2014.01.012) Copyright © 2014 National Kidney Foundation, Inc. Terms and Conditions