Volume 9, Issue 4, Pages 207-214 (September 2003) Attenuation of homocysteine-induced endothelial dysfunction by exercise training Reid Hayward, Ratree Ruangthai, Peter Karnilaw, Adam Chicco, Robert Strange, Heidi McCarty, Kim C. Westerlind Pathophysiology Volume 9, Issue 4, Pages 207-214 (September 2003) DOI: 10.1016/S0928-4680(03)00023-3
Fig. 1 Vasoreactive response of aortic rings from SED and EX rats to endothelium-dependent (ACh, A-23187) and endothelium-independent vasodilators following exposure to HCY. *P<0.05 vs. SED+HCY. Pathophysiology 2003 9, 207-214DOI: (10.1016/S0928-4680(03)00023-3)
Fig. 2 Aortic eNOS protein content in SED and EX rats at baseline and following 6 weeks of exercise training. *P<0.05 vs. SED 6 weeks. Pathophysiology 2003 9, 207-214DOI: (10.1016/S0928-4680(03)00023-3)
Fig. 3 Aortic eNOS activity based on the conversion of l-arginine to l-citrulline in pooled samples taken from SED and EX animals following 6 weeks of exercise training. Pathophysiology 2003 9, 207-214DOI: (10.1016/S0928-4680(03)00023-3)