Eight-year experience with cryopreserved arterial homografts for the in situ reconstruction of abdominal aortic infections Theodosios Bisdas, MD, Martin.

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Presentation transcript:

Eight-year experience with cryopreserved arterial homografts for the in situ reconstruction of abdominal aortic infections Theodosios Bisdas, MD, Martin Bredt, MD, Maximilian Pichlmaier, MD, Thomas Aper, MD, Mathias Wilhelmi, MD, Sotirios Bisdas, MD, Axel Haverich, MD, Omke E. Teebken, MD Journal of Vascular Surgery Volume 52, Issue 2, Pages 323-330 (August 2010) DOI: 10.1016/j.jvs.2010.02.277 Copyright © 2010 Society for Vascular Surgery Terms and Conditions

Fig 1 Levels for C-reactive protein (CRP), leucocytes, and platelets during the first 10 postoperative days (POD) are shown in 57 patients undergoing cryopreserved arterial homografts implantation in the abdominal aorta due to a vascular infection. Journal of Vascular Surgery 2010 52, 323-330DOI: (10.1016/j.jvs.2010.02.277) Copyright © 2010 Society for Vascular Surgery Terms and Conditions

Fig 2 Kaplan-Meier life-table analysis presents (A) survival rates and (B) freedom from reoperation rates after cryopreserved arterial homografts replacement against vascular infections in the abdominal aorta. The 95% confidence interval is shown on both figures. Journal of Vascular Surgery 2010 52, 323-330DOI: (10.1016/j.jvs.2010.02.277) Copyright © 2010 Society for Vascular Surgery Terms and Conditions

Fig 3 A, Histologic examination of the arterial wall from cryopreserved arterial homografts (CAH) before implantation show slight edema of tunica intima and fibrosis (1), but the internal elastic membrane is identifiable (2) as a thick line between the tunica intima and media). The tunica media has low number of elastic fibers, and the smooth muscle cells (SMCs) with their cores are arranged in a normal spiral orientation around the long axis of the vessel (Alcian blue stain, original magnification ×100). B, Histologic examination of a degenerated CAH 2 years after implantation shows intraluminal thrombus (red material, 1), edema, and fibrosis of the tunica intima, as well as atherosclerosis (foamy cells, 2). Neither internal elastic membrane nor SMCs of the media are identifiable, and multifocal destruction of the tunica media and elastic fibers (not shown) has occurred (hematoxylin and eosin stain; original magnification ×100). Journal of Vascular Surgery 2010 52, 323-330DOI: (10.1016/j.jvs.2010.02.277) Copyright © 2010 Society for Vascular Surgery Terms and Conditions