Normal ECG waves & ARRYHTHMIAS

Slides:



Advertisements
Similar presentations
EKG Review.
Advertisements

ECG TRAINING MODULE 4 BY BRAD CHAPMAN RCT.
ECG Rhythm Interpretation
By Dr.Ahmed Mostafa Assist. Prof. of anesthesia & I.C.U.
The electrocardiogram (ECG or EKG)
Understanding Cardiac Electrophysiology
“ Heart Blocks”.
ECG Rhythm Interpretation
ECG Rhythm Interpretation
Portland Community College
Chapter 5 Atrial Rhythms
Heart Arrhythmia's Brandy Parker Brianne Negen Jeremy Grimm
Normal sinus rhythm Debs Farr 2011.
Fast & Easy ECGs, 2nd E – A Self-Paced Learning Program
Cardiovascular System Block Cardiac Arrhythmias (Physiology)
Arrythmia Interpretation (cont’d) Rates of automaticity – Too fast (tachycardia) – Too slow (bradycardia) – Too irritable (Premature) – Absent (block)
Basic Dysrhythmia &Recording ECG
Natalia Fernandez, PT, MS, MSc, CCS University of Michigan Health Care System Department of Physical Medicine and Rehabilitation.
Electrocardiogram Primer (EKG-ECG)
Cardiovascular Monitoring Electrocardiogram
EKG Interpretation: Arrhythmias Humayun J. Chaudhry, D.O., FACP, FACOI Assistant Dean for Pre-Clinical Education and Chairman, Department of Medicine N.Y.
Guide For Arrhythmia Recognition
Department faculty and hospital therapy of medical faculty and department internal diseases of medical prophylactic faculty. Cardiac arrhythmia Docent.
Fast & Easy ECGs – A Self-Paced Learning Program
Disease of Cardiac System
EKG Interpretation.
Abnormal Sinus Rhythms
Lecture Objectives Describe sinus arrhythmias Describe the main pathophysiological causes of cardiac arrhythmias Explain the mechanism of cardiac block.
Normal electrocardiogram
EKG Interpretation: Arrhythmias Mustafa Salehmohamed, D.O. Assistant Clinical Instructor Department of Medicine N.Y. College of Osteopathic Medicine October.
Copyright ©2010 by Pearson Education, Inc. Upper Saddle River, New Jersey All rights reserved. Understanding EKGs: A Practical Approach, Third Edition.
ECG interpretations.
Junctional Dysrhythmias
Cardiac Conduction. Physiology of Cardiac Conduction The excitatory & electrical conduction system of the heart is responsible for the contraction and.
For more presentations FOR MORE FREE MEDICAL POWERPOINT PRESENTATIONS VISIT WEBSITE blogspot.com.
Fast & Easy ECGs – A Self-Paced Learning Program
Adel Hasanin, MRCP (UK), MS (Cardiology)
Chapters 11, 12, 13 Electrocardiogram Dr. Marko Ljubković Department of Physiology.
Normal EKG – P wave: Atrial depolarization – PR interval: < 0.20 sec – QRS complex: ventricular depolarization – QRS interval < 0.10 sec SA 0.10 – 0.12.
Fast & Easy ECGs, 2nd E – A Self-Paced Learning Program
Gail Walraven, Basic Arrhythmias, Sixth Edition ©2006 by Pearson Education, Inc., Upper Saddle River, NJ Chapter 7 Heart Blocks.
ECG intereptation Abdualrahman ALshehri Lecturer King Saud University
 Any atrial area may originate an impulse.  Rhythms have upright P waves preceding each QRS complex.  Not as well-rounded  Heart rates usually from.
EKG’s Kelly Marchant RN July 28, 2015 Adapted from NURO 438
MECHANISMS OF CARDIAC ARRHYTHMIAS. DR AMNA TAHIR PHYSIOLOGY DEPARTMENT. KEMU.
Chapter 2 - ECG Supraventricular Rythms Early beats arising from above the ventricles. PAC, PJC.
Q I A 6 Fast & Easy ECGs – A Self-Paced Learning Program QRS Complexes.
ECG Rhythm Interpretation Module I ECG Basics Share what you know, learn what you don’t.
Electrocardiography – Abnormalities (Arrhythmias) 7
Introduction to the EKG. Electricity of the Heart The contraction of any muscle is associated with electrical changes called depolarizations and can be.
Fast & Easy ECGs – A Self-Paced Learning Program
1 © 2013 The McGraw-Hill Companies, Inc. All rights reserved. Fast & Easy ECGs, 2E P Waves Fast & Easy ECGs, 2nd E – A Self- Paced Learning Program 66.
ARRHYTHMIA Objectives At the end of this session students should be able to:  Distinguish the normal from abnormal rhythms.  Understand the pathophysiologic.
Steps in Rhythm Analysis Evaluation of ECG requires systematic approach to analyzing given rhythm –Numerous methods can be used for rhythm interpretation.
Lecture Objectives Describe sinus arrhythmias Describe the main pathophysiological causes of cardiac arrhythmias Explain the mechanism of cardiac block.
Fast & Easy ECGs – A Self-Paced Learning Program
Chapter 2 - ECG Supraventricular Rythms Early beats arising from above the ventricles. PAC, PJC.
Chapter 5. Remember…  If sinus node loses its pacemaking role for whatever reason, the next fastest site will take over.  Rhythms that start in the.
Arrhythmias and EKGs.
8 Introducing the Atrial Rhythms 1.
Heart Blocks Leaugeay Webre BS, CCEMT-P, NREMT-P.
Atrial Rhythms MEHDI BAKHSHI MSN CCN ICNS. Wandering Pacemaker Pacemaker site wanders between sinus node, atria, and AV junction. Rate is usually normal;
ECG RHYTHM ABNORMALITIES
Atrial and Ventricular Arrhythmias
Objective 12 Electrocardiograms
Chapter 4 Atrial Rhythms.
Cardiovascular System Block Cardiac Arrhythmias (Physiology)
Antiarrhythmic drugs [,æntiə'riðmik] 抗心律失常药
ECG Rhythm Interpretation
Presentation transcript:

Normal ECG waves & ARRYHTHMIAS

P wave & P-R interval The P wave represents the wave of depolarization that spreads from the SA node throughout the atria, and is usually 0.08 to 0.1 seconds (80-100 ms) in duration. The brief isoelectric (zero voltage) period after the P wave represents the time in which the impulse is traveling within the AV node (where the conduction velocity is greatly retarded) and the bundle of His. Atrial rate can be calculated by determining the time interval between P waves. Click here to see how atrial rate is calculated. The period of time from the onset of the P wave to the beginning of the QRS complex is termed the P-R interval, which normally ranges from 0.12 to 0.20 seconds in duration. This interval represents the time between the onset of atrial depolarization and the onset of ventricular depolarization. If the P-R interval is >0.2 sec, there is an AV conduction block, which is also termed a first-degree heart block if the impulse is still able to be conducted into the ventricles.

QRS complex The QRS complex represents ventricular depolarization. Ventricular rate can be calculated by determining the time interval between QRS complexes. The duration of the QRS complex is normally 0.06 to 0.1 seconds. This relatively short duration indicates that ventricular depolarization normally occurs very rapidly. If the QRS complex is prolonged (> 0.1 sec), conduction is impaired within the ventricles. This can occur with bundle branch blocks or whenever a ventricular foci (abnormal pacemaker site) becomes the pacemaker driving the ventricle. Such an ectopic foci nearly always results in impulses being conducted over slower pathways within the heart, thereby increasing the time for depolarization and the duration of the QRS complex.

QRS-Complex

T wave The T wave represents ventricular repolarization and is longer in duration than depolarization (i.e., conduction of the repolarization wave is slower than the wave of depolarization). Sometimes a small positive U wave may be seen following the T wave (not shown in figure at top of page). This wave represents the last remnants of ventricular repolarization. Inverted or prominent U waves indicates underlying pathology or conditions affecting repolarization.

Q-T interval The Q-T interval represents the time for both ventricular depolarization and repolarization to occur, and therefore roughly estimates the duration of an average ventricular action potential. This interval can range from 0.2 to 0.4 seconds depending upon heart rate. At high heart rates, ventricular action potentials shorten in duration, which decreases the Q-T interval. Because prolonged Q-T intervals can be diagnostic for susceptibility to certain types of tachyarrhythmias, it is important to determine if a given Q-T interval is excessively long. In practice, the Q-T interval is expressed as a "corrected Q-T (QTc)" by taking the Q-T interval and dividing it by the square root of the R-R interval (interval between ventricular depolarizations). This allows an assessment of the Q-T interval that is independent of heart rate. Normal corrected Q-Tc intervals are less than 0.44 seconds.

General Terms: Normal sinus rhythm - heart rhythm controlled by sinus node at a rate of 60-100 beats/min; each P wave followed by QRS and each QRS preceded by a P wave. Bradycardia - a heart rate that is lower than normal. Tachycardia - a heart rate that is higher than normal. Paroxysmal - an arrhythmia that suddenly begins and ends.

Sinus Arrhythmias Sinus bradycardia - low sinus rate <60 beats/min. Sinus tachycardia - high sinus rate of 100-180 beats/min as occurs during exercise or other conditions that lead to increased SA nodal firing rate. Sick sinus syndrome - a disturbance of SA nodal function that results in a markedly variable rhythm (cycles of bradycardia and tachycardia.

Paroxysmal Atrial Tachycardia Atrial tachycardia - a series of 3 or more consecutive atrial premature beats occurring at a frequency >100/min; usually due to abnormal focus within the atria and paroxysmal in nature, therefore appearance of P wave is altered in different ECG leads.  This type of rhythm includes paroxysmal atrial tachycardia (PAT. (

Supraventricular tachycardia (SVT) usually caused by reentry currents within the atria or between ventricles and atria producing high heart rates of 140-250; the QRS complex is usually normal width, unless there are also intraventricular conduction blocks (e.g., bundle branch block

Ventricular premature Contractions (VPCs(: caused by ectopic ventricular foci; characterized by widened QRS; often referred to as a premature ventricular complex, or PVC.

Atrial arrhythmias Atrial flutter - sinus rate of 250-350 beats/min. .

Ventricular tachycardia (VT) - high ventricular rate caused by aberrant ventricular automaticity (ventricular foci) or by intraventricular reentry; can be sustained or non-sustained (paroxysmal); usually characterized by widened QRS (>0.14 sec); rates of 100 to 280 beats/min; life-threatening. Ventricular fibrillation - uncoordinated ventricular depolarizations; leads to death if not quickly converted to a normal rhythm or at least a rhythm compatible with life.

AV nodal blocks Conduction block within the AV node (or occasionally in the bundle of His) that impairs impulse conduction from the atria to the ventricles.

First-degree AV nodal block the conduction velocity is slowed so that the P-R interval is increased to greater than 0.2 seconds.  Can be caused by enhanced vagal tone, digitalis, beta-blockers, calcium channel blockers, or ischemic damage.

Second-degree AV nodal block The conduction velocity is slowed to the point where some impulses from the atria cannot pass through the AV node.  This can result in P waves that are not followed by QRS complexes.  For example, 1 or 2  P waves may occur alone before one is followed by a QRS.  When the QRS follows the P wave, the P-R interval is increased.  In this type of block, the ventricular rhythm will be less than the sinus rhythm.

Third-degree AV nodal block A conduction through the AV node is completely blocked so that no impulses are able to be transmitted from the atria to the ventricles.  QRS complexes will still occur (escape rhythm), but they will originate from within the AV node, bundle of His, or other ventricular regions.  Therefore, QRS complexes will not be preceded by P waves.  Furthermore, there will be complete asynchrony between the P wave and QRS complexes.  Atrial rhythm may be completely normal, but ventricular rhythm will be greatly reduced depending upon the location of the site generating the ventricular impulse.  Ventricular rate typically range from 30 to 40 beats/min.

Atrial fibrillation there are no P waves present instead of P waves, fibrillarory "F" waves with a rate higher than 350 per minute are present the interval between QRS complexes is variable and there is no apparent rule in the rhythm of the QRS complexes (absolute arrhythmia) depending on the frequency of the QRS complexes we distinguish atrial fibrillation with bradycardic (less than 60 per minute), normocardic (60 to 100 per minute) and tachycardic (over 100 per minute) response of the ventricles uncoordinated atrial depolarizations