Secondary oxalosis due to excess vitamin C intake

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Secondary oxalosis due to excess vitamin C intake S.H. Nasr, Y. Kashtanova, V. Levchuk, G.S. Markowitz Kidney International Volume 70, Issue 10, (November 2006) DOI: 10.1038/sj.ki.5001724 Copyright © 2006 International Society of Nephrology Terms and Conditions

Figure 1 Proximal tubules display diffuse degenerative changes characterized by luminal ectasia, epithelial simplification with coarse cytoplasmic vacuolization, and irregular luminal contours. Mild interstitial edema is present. The findings are also notable for intratubular deposits of calcium oxalate (arrows). (Hematoxylin and eosin; original magnification, × 400.) Kidney International 2006 70, DOI: (10.1038/sj.ki.5001724) Copyright © 2006 International Society of Nephrology Terms and Conditions

Figure 2 When viewed under polarized light, the tubular calcium oxalate deposits appear birefringent. (Hematoxylin and eosin; original magnification, × 200.) Kidney International 2006 70, DOI: (10.1038/sj.ki.5001724) Copyright © 2006 International Society of Nephrology Terms and Conditions