Dilja D. Krueger, Nils Brose Neuron

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Presentation transcript:

Evidence for a Common Endocannabinoid-Related Pathomechanism in Autism Spectrum Disorders Dilja D. Krueger, Nils Brose Neuron Volume 78, Issue 3, Pages 408-410 (May 2013) DOI: 10.1016/j.neuron.2013.04.030 Copyright © 2013 Elsevier Inc. Terms and Conditions

Figure 1 Potential Role of Nlgn3 at Perisomatic Inhibitory Synapses on CA1 Pyramidal Neurons PV and CCK basket cells form perisomatic inhibitory synapses onto pyramidal neurons. Boxed inserts summarize the data reported by Földy et al. (2013). Expanded views show a model of how Nlgn3 may affect PV basket cell synapses (upper left) and CCK basket cell synapses (lower right) based on these findings. It should be noted that the presence of Nlgn3 at these synapses has not been investigated and is inferred from the data. Abbreviations: Nlgn3, Neuroligin-3; PV, parvalbumin; CCK, cholecystokinin; SV, synaptic vesicle; MOR, μ-opioid receptor; M2, M2 muscarinic acetylcholine receptor; CB1R, cannabinoid receptor type 1. Neuron 2013 78, 408-410DOI: (10.1016/j.neuron.2013.04.030) Copyright © 2013 Elsevier Inc. Terms and Conditions