Bryan Bjornstad, MD, Scott H. Goodman, MD, Joseph I

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Presentation transcript:

Paroxysmal Sleep as a Presenting Symptom of Bilateral Paramedian Thalamic Infarctions Bryan Bjornstad, MD, Scott H. Goodman, MD, Joseph I. Sirven, MD, David W. Dodick, MD Mayo Clinic Proceedings Volume 78, Issue 3, Pages 347-349 (March 2003) DOI: 10.4065/78.3.347 Copyright © 2003 Mayo Foundation for Medical Education and Research Terms and Conditions

Figure 1 Electroencephalogram (EEG) obtained during the patient's waking state as patient responded appropriately to questions (sensitivity = 7 µV/mm, high-frequency filter = 35 cycles/second, low-frequency filter = 0.53 cycles/second). The EEG is characterized by theta frequency slowing and muscle artifact. Mayo Clinic Proceedings 2003 78, 347-349DOI: (10.4065/78.3.347) Copyright © 2003 Mayo Foundation for Medical Education and Research Terms and Conditions

Figure 2 Electroencephalogram (EEG) obtained 9 minutes after Figure 1 EEG during the patient's episode of unresponsiveness. Note the 14-Hz sleep spindles prominent in the central channels (arrows) (sensitivity = 5 µV/mm, high-frequency filter = 35 cycles/second, low-frequency filter = 0.53 cycles/second) and the absence of muscle artifact. Mayo Clinic Proceedings 2003 78, 347-349DOI: (10.4065/78.3.347) Copyright © 2003 Mayo Foundation for Medical Education and Research Terms and Conditions

Figure 3 Axial diffusion-weighted magnetic resonance image of patient's head shows increased signal in the paramedian thalamic nuclei bilaterally. considered in patients who present with recurrent episodes of unresponsiveness. Mayo Clinic Proceedings 2003 78, 347-349DOI: (10.4065/78.3.347) Copyright © 2003 Mayo Foundation for Medical Education and Research Terms and Conditions