Fig. 6. Quantitation of hntXE81 loss of function marked clones induced in AMPs or ISCs using ISC/EB (esgGAL4) and EC (NP6293) specific reporters. Quantitation.

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Volume 8, Issue 1, Pages (January 2011)
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Drosophila piwi Mutants Exhibit Germline Stem Cell Tumors that Are Sustained by Elevated Dpp Signaling  Zhigang Jin, Alex S. Flynt, Eric C. Lai  Current.
Intestinal Stem Cell Pool Regulation in Drosophila
Differences in cellular organization emerge from quantitative proteomic experiments Differences in cellular organization emerge from quantitative proteomic.
In Vivo Analysis of Centromeric Proteins Reveals a Stem Cell-Specific Asymmetry and an Essential Role in Differentiated, Non-proliferating Cells  Ana.
A Sox Transcription Factor Is a Critical Regulator of Adult Stem Cell Proliferation in the Drosophila Intestine  Fanju W. Meng, Benoît Biteau  Cell Reports 
Benoît Biteau, Heinrich Jasper  Cell Reports 
KIN-3 depletion leads to increased levels of centrosomal ZYG-1
Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.
Fig. 4. Clinically defined SMN mutants show alterations in the association with telomerase complex proteins. Clinically defined SMN mutants show alterations.
Fig. 3. Hts and Dlg are in a complex at the postsynaptic membrane of larval NMJs.(A–B″) PLA with HtsM and Dlg antibodies (green) performed on third instar.
Fig. 4. E-cadherin expression level affects monomer dynamics.
Fig. 7. Motion adaptation increases time-dependent response modulations (TDRM) relatively to the average cell response.TDRM normalized to the value obtained.
Fig. 2. Proportion of motile objects and track length
Total and differential cell counting in the broncho-alveolar lavage of mice exposed to CS or air during 3 or 6 months. Total and differential cell counting.
Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle
Fig. 4. A primary screen based on scrape closure
Fig. 4. Effect of royal jelly on silkmoth fat body cells and eggs.
The effects of a dominant negative mutant of lamin B1 on lamin distribution in HeLa cells. The effects of a dominant negative mutant of lamin B1 on lamin.
Fig. 3. Inactivation of the Wnt/β-catenin signaling pathway inhibited cell proliferation and induced apoptosis in A549 and SPC-A-1 cells. Inactivation.
Fig. 1. Morphological and growth characterization of hBMCs and hPDCs
Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.
Fig. 4. Mutations induced by TALENs are heritable through the germline
eb1a-2 eb1b-3 double-mutant plants display skewed and shorter roots
Fig. 3. Genetic interactions between unc-53 and unc-6 and between unc-53 and unc-5 affect intracellular UNC-40::GFP localization.(A–H) Photomicrographs.
Bgs1p does not localize to the contractile ring during or after mitosis in the septation initiation network mutants cdc and cdc Bgs1p does.
Fig. 4. Targeted disruption of STK35 transcripts in mouse.
Fig. 2. Two signal-producing behaviours of wild-type Canton-S males and per mutant males relative to whether the wild-type female is moving or immobile.
Table 3. Penetrance of cuticle defects seen in ush2 mutants embryos and in the progeny from rescue experiments (genotypes indicated in the table) with.
Fig. 6. Comparison of Plk4 with Sas-6 localization
Fig. 3. The checkpoint proteins Mad2 and BubR1 remain associated with the kinetochores of unaligned chromosomes in cenp-metaΔ mutant cells entering anaphase.(A–C)
The TER94-p47 complex isinvolved in Notch signaling regulation
Fig. 10. A mutant clone homozygous for mip120LL07629 has greatly diminished Mip120 protein levels. hsFLP/+; FRT42B, mip120LL07629/FRT42B, Ubi-GFP-nls females.
Fig. 7. Representative images of control (Cas9+GFP) and Cas9+gRNA+GFP co-injected embryos on day 4 of culture, showing nuclear-imported GFP (green) and.
Fig. 7. Ror-GFP binds to Myc-tagged Wnts and Wnt receptors.
Elevated expression of vegfaa in hemangiosarcoma.
Delayed formation of sensory hair cells in dlx3b/4b-deficient embryos
Bivariate plots of mean total scratches and mean total pits for natural grazers. Bivariate plots of mean total scratches and mean total pits for natural.
Effects of the ft-overexpressing clones in A and P compartments (cf.
Fig. 6. Apical polarity of primitive endoderm cells on surface of embryoid bodies.Embryoid bodies were analyzed by immunofluorescence microscopy for GATA4.
Fig. 8. The morphology of the ventral nerve cord in Ror-Myc overexpressing embryos is normal. The morphology of the ventral nerve cord in Ror-Myc overexpressing.
Fig. 3. Overexpression of wild-type GFP-CPAP, but not GFP-CPAP-377EE (GFP-377EE), induces cilia formation and promotes the growth of cilia.CAD cells (A,B)
Fig. 1. Polarized F-actin cables in the Xenopus neural plate.
BAF60c transcriptionally affects cardiac morphogenesis and function
Depleting CK2 restores centrosomal ZYG-1 levels in zyg-1(it25) embryos
Delay of tail resorption in trβ crispants during natural metamorphosis
Loss of Graf results in plasmatocyte overproliferation.
Morphological changes induced by T3 treatment in trβ crispants
Fig. 3. transparent is required cell-autonomously in iridophores
Fig. 2. Expression of Cx43 mutant T154A resulted in non-radial spreading and formation of protrusions in J558µm3 cells spreading in response to BCR signaling.(A)
Peroxisome speeds were slower in patient and control cells.
Fig. 2. Acetylation stiffens primary cilia.
Fig. 1. Cell adhesion molecule expression and the aggregation of wildtype and mutant ES cells.(A) Wildtype (WT), E-cadherin null (9J), and N-cadherin null.
Fig. 8. Compared mobilities of passenger proteins in G2/M-prophase, metaphase and anaphase.FRAP experiments were performed on HeLa cells stably expressing.
Fig. 1. Rnd2 and Rnd3 induce stress fibres whereas Rnd1 reduces stress fibres in endothelial cells.(A) Rnd mRNAs are expressed in HUVECs. Total RNA was.
Fig. 3. MO-mediated smc3 knockdown results in reduced regenerate length, segment length and cell proliferation. MO-mediated smc3 knockdown results in reduced.
Fig. 3. Mean force and velocity during jumping
Fig. 5. EGL-20 inhibits anterior and posterior orientation of UNC-40 asymmetric localization and the formation of axons from these sites.(A–D) HSN neurons.
mip120 null egg chambers have a condensed nurse cell DNA phenotype
Fig. 6. Bj mutants show stereocilia patterning defects and biliary duct (BD) malformations. Bj mutants show stereocilia patterning defects and biliary.
Kinetics of BDNF-induced Erk, Akt and PLCγ activation in the presence of 15 mM NaCl or 15 mM KCl. Representative western blots (A) and quantitative plots.
Fig. 1. Microarray analyses of genes whose expression is regulated by innervation during synaptogenesis.(A) Schematic drawings of the experimental design.
Fig. 5. Behaviours of the wild-types Oregon-R at two temperatures.
Fig. 3. Inclusion of E-cadherin into stationary clusters requires cis-, trans-, and cytoplasmic interactions. Inclusion of E-cadherin into stationary clusters.
Failure of chromosome disassembly in mip120 null ovarian nurse cells
Fig. 7. Analysis of dFMRP kinetics in dFMRP granules by FRAP
Fig. 1. Effects of royal jelly on silkmoth body size.
Decreased enzymatic activity in PKG mutants increases time to recovery from an electroshock. Decreased enzymatic activity in PKG mutants increases time.
Genetic interaction between e(y)1 and Notch in wing development.
Quantitative analysis of effects of the Egr inhibitor construct on neurite outgrowth. Quantitative analysis of effects of the Egr inhibitor construct on.
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Fig. 6. Quantitation of hntXE81 loss of function marked clones induced in AMPs or ISCs using ISC/EB (esgGAL4) and EC (NP6293) specific reporters. Quantitation of hntXE81 loss of function marked clones induced in AMPs or ISCs using ISC/EB (esgGAL4) and EC (NP6293) specific reporters. (A-D) Box plot diagrams are shown displaying the distribution GFP-positive cell counts using esg-MARCM (A,C) or NP6293-MARCM (B,D) to indicate adult ISC/EB and EC fates, respectively. The propensity of hntXE81 mutant cells to be recovered as ISCs/EBs following clone induction in ISCs (adult stage) is not markedly different from the control (A), whereas the propensity of hntXE81 mutant cells to differentiate as ECs following clone induction in adult ISCs is greatly reduced (B). Following clone induction in AMPs (during third instar larval stage) hntXE81 mutant cells are never recovered as ISCs/EBs in the adult midgut (C) whereas the same conditions result in ample hntXE81 mutant ECs (D). The median value is shown by a horizontal black line within each box. The median value for the recovery of hntXE81 mutant NP6293-MARCM positive cells following ISC clone induction (B) is zero (14/23 midguts with no GFP-positive cells). The median value for the recovery of hntXE81 mutant esg-MARCM positive cells following AMP clone induction (C) was also zero (23/23 midguts with no GFP-positive cells). Brittany L. Baechler et al. Biology Open 2016;5:1-10 © 2016. Published by The Company of Biologists Ltd